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Inhibition of LINC02163 suppressed cell proliferation, while transfection of miR-511-3p inhibitor or AKT3 in LINC02163-depletion cells restored cell growth and abolished the cell cycle arrest in G0/G1 phase. Therefore, it was indicated that LINC02163 exerted pro-tumour effect through miR-511-3p...
miR-511-3p, embedded in the macrophagemannose receptor gene, contributes to intestinalinflammationSEMHeinsbroek 1 ,MLSquadrito 2 ,RSchilderink 1 ,FWHilbers 1 ,CVerseijden 1 ,MHofmann 1 ,AHelmke 1 ,L Boon 3 , ME Wildenberg 1 , JJTH Roelofs 4 , CY Ponsioen 1 , CP Peters 1 , AA te...
NF-κB p50 siRNA是一种小干扰RNA,用于特异性抑制NF-κB p50亚单位的表达,调控炎症反应和免疫反应;miR-511-3p是一种microRNA,能够通过靶向特定基因调控细胞增殖、迁移及抗肿瘤作用。IL4R结合肽IL4RPep-1修饰负载NF-κB p50 siRNA和miR-511-3p的巨噬细胞外泌体是一种具有高度靶向性和免疫调节作用的治疗载体。
目的 研究LncRNA OIP5-AS1对肺癌细胞的增殖,迁移和侵袭的影响以及作用机制.方法 采用qRT-PCR法检测OIP5-AS1和miR-511-3p的表达量;采用MTT法检测细胞增殖;采用Transwell小室实验检测细胞迁移和侵袭;蛋白免疫印迹法检测CyclinD1,MMP-2蛋白表达量;双荧光素酶报告实验检测OIP5-AS1和miR-511-3p的靶向关系.结果 与正常...
miR‐511‐3pZFPM2‐AS1Lung cancer is the dominating cause of cancer‐induced death and can be classified into small cell lung cancer and non–small cell lung cancer (NSCLC). Lung adenocarcinoma (LUAD) is the most common histological subtype of NSCLC and its pathology remains unclear. Mounting...
Blot法检测CVA患者血浆样本中miR-511-3p的转录水平.结果通过miR-511-3p转染发现,miR-511-3p过表达时,M1型巨噬细胞中TNF-α表达减少,CCL17表达增多(P <0.05);CVA小鼠模型中TNF-α和IRF3磷酸化水平减少,TGF-β增多(P <0.05);同时与非哮喘患者相比,CVA患者miR-511-3p表达减少(P <0.05).结论 miR-511-3p可...
MiR-511-3p is embedded in intron 5 of the CD206/MRC1 geneMrc1, expressed by macrophage and dendritic cell populations. CD206 and miR-511-3p expression are co-regulated, and their contribution to intestinal inflammation is unclear. We investigated their roles in intestinal inflammation in both ...
Moreover, ZFPM2-AS1 functioned as a molecular sponge of miR-511-3p, which was a suppressor in LUAD. Moreover, ZFPM2-AS1 sponged miR-511-3p and thereby deregulated AF4/FMR2 family member 4 (AFF4), a target of miR-511-3p. At length, rescue assays indicated that AFF4 overexpression ...
However, the role of miR-511-3p within the AD-MSCs -derived EV in SCI is largely unknown. Western blotting results demonstrated the secretion of EVs derived from AD-MSCs under hypoxia (Hyp-EVs) was more than those under normoxia (Nor-EVs), and miR-511-3p expression was more enriched in...