Cis and trans acting factors involved in human cytomegalovirus experimental and natural latent infection of CD14 (+) monocytes and CD34 (+) cells. PLoS Pathog. 2013, 9, e1003366. [CrossRef] 122. Margueron, R.; Reinberg, D. The Polycomb complex PRC2 and its mark in life....
(TLR/MyD88) and NK cell receptor protein 1 (NKR-P1) for chitosan, CD14, CD18, TLR2, TLR4 for alginate, α5β1 integrin for fibroin, HA-mediated motility (RHAMM), a cluster of differentiation-44 (CD44) for hyaluronan, endothelial cell receptor ICAM-1 and the leukocyte receptor Mac-1 ...
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CCL18 is mainly expressed by lung and some lymphoid tissues like lymph nodes express CCL18 at low level. It is chemotactic for both activated (CD3+) T cells and nonactivated (CD14-) lymphocytes, but not for monocytes or granulocytes. Involved in B-cell migration into B-cell follicles in ...
抑制NK细胞的杀伤免疫应答MSC抑制NK细胞增殖MSC与NK细胞 MSC能抑制IL-2刺激的NK细胞IFN-γ分泌 MSC本身不引起NK细胞的IFN-γ分泌静止及活化的DC的形态特征ActivatedDendriticcellsexpressB7moleculesImmatureDCMatureDCLangerhans’CellMigrationAntigenFromJaneway,CAImmunobiology,5thed.骨髓DC前体血流非淋巴组织分化非成熟DC...
Maatouk 等[27] 发现,热处理 后的柚皮素能够增强小鼠 NK 细胞的杀伤活性,并 抑制 T 细胞的细胞毒作用. 3.2 黄酮类化合物对细胞因子的影响 细胞因子是由免疫细胞经刺激合成,分泌的 一类具有广泛生物学活性的小分子蛋白质. Mori⁃ moto 等[28] 采用实时荧光定量 PCR 方法检 测口服 芦丁 10 d 的 C3H / ...
Human NK cells are defined by the phenotype CD3 − CD56 + ; additionally they are CD19 and CD14 negative. The only marker that is specific for NK cells is NKp46. NK cells comprise 5–15% of all circulating lymphocytes (Lanier et al., 1986; Walzer et al., 2007). They are ...
[17].在 标准 培养 条件下 ,可 表达 细胞 表面 标记 CD105,CD73 和CD90,而不表达细胞表面标记CD45, CD34,CD14或CD11b,CD79,CD19或HLA-DR[18], 其具有自我更新,多向分化潜能,可体外大量扩增等干 细胞生物学特性[19-21].心肌梗死后移植骨髓间充质干细 胞能够增加新生血管数量,减小梗死面积,减少瘢痕[...
Many immune cells express dopamine receptors and other dopamine related proteins, enabling them to actively respond to dopamine and suggesting that dopaminergic immunoregulation is an important part of proper immune function. A detailed understanding of the physiological concentrations of dopamine in specific...
cells and monocytes by changing the pattern of miRNA expression, and indeed, this may be part of the mechanism underpinning this phenomenon. The capacity of NAT therapy to inhibit the expression of HERV-W/MSRV in B cells, monocytes and NK cells in patients with RRMS without any changes in ...