glutamine synthetaseα -fetoproteinhepatomachemical carcinogenesisThe activities of glutaminase and glutamine synthetase were determined in rats during induction of carcinogenesis by azo-dye. In the hepatoma tissue, the activities of phosphate-dependent glutaminase (PDG) and phosphate-independent glutaminase (...
G0199 谷氨酰胺试剂盒(Glutamine) 酶法 48T/96T 800/1500 G0200 谷氨酰胺合成酶试剂盒(Glutamine synthetase) 酶法 48T/96T 800/1500 G0210 谷胱甘肽还原酶试剂盒(glutathion reductases) 酶法 48T/96T 800/1500 G0211 谷胱甘肽试剂盒(Glutathione ) 酶法 48T/96T 800/1500 G0212 谷胱甘肽还原酶活性系数(GRAC...
Pediatric ResearchDeMarco, V., McCain, M., Strauss, D., Shenoy, V., Chakrabarti, R. & Neu, J. (1996) Glutamine synthetase and glutaminase in the human placenta: clues to a glutamine-glutamate shuttle? Pediatric Research, 39, 308A....
On release, its action is terminated by its being taken up into glial cells via anexcitatory amino acid transporter(EAAT) and then converted to glutamine byglutamine synthetase. Glutamine transporters then move the glutamine from the glial cell into the neuron (Siegel et al., 2006;Stahl, 2013...
This study examined the effect of starvation on the activity, level of mRNA, and distribution of mRNA of glutaminase and glutamine synthetase in the rat intestine. Rats were randomized into groups and were either: (1) fed for 2 days with rat food ad libitum or (2) starved for 2 days. ...
Glutamine Synthetase Antibody (YA405) Glutathione Reductase Antibody Transglutaminase 2 Antibody (YA2255) 体内研究 (In Vivo) BPTES -NP (BPTES 纳米颗粒,100 μL 纳米颗粒中含有 1.2 mg BPTES,静脉注射) 显著减弱患者来源的胰腺原位肿瘤模型中的肿瘤生长[1]。
Watford, M., and G. Wu. 2005. Glutamine metabolism in urico- telic species: Variation in skeletal muscle glutamine synthetase, glutaminase, glutamine levels and rates of protein synthesis. Comp. Biochem. Physiol. 140B:607-614.Watford M, Wu G (2005) Glutamine metabolism in uricotelic species:...
Many transformed cells exhibit altered glucose metabolism and increased utilization of glutamine for anabolic and bioenergetic processes. These metabolic adaptations, which accompany tumorigenesis, are driven by oncogenic signals. Here we report that the
metabolic intermediates3. L-Glutaminase has received significant attention since it was reported extensively as antileukemic agent4-6. Unlike normal cells, leukemic cell do not demonstrate the L-glutamine synthetase, thus it is dependent on the exogenous supply of L-glutamine for their growth ...
The glutaminase enzymes GAC and GLS2 catalyze the hydrolysis of glutamine to glutamate, satisfying the ‘glutamine addiction’ of cancer cells. They are the targets of anti-cancer drugs; however, their mechanisms of activation and catalytic activity have