L.; BLUETHMANN, H.; RAIVICH, G.: Interleukin-6 (IL6) and cellular response to facial nerve injury: effects on lymphocyte recruitment, early microglial activation and axonal outgrowth in IL6- deficient mice. Eur. J. Neurosci., 14(2):327-341, Jul. 2001...
Interleukin-6 alters the cellular responsiveness to clopidogrel, irinotecan, and Oseltamivir by 维普资讯 http://www.cqvip.com
Key elements of the SASP are the proinflammatory cytokines interleukin-6 (IL-6), interleukin-8 (IL-8), and interleukin-1(IL-1)「 IL-1is considered one of the master regulators of the SASP 」 in OIS: chemokines binding to the IL-8 receptor C-X-C motif chemokine receptor 2...
Key elements of the SASP are the proinflammatory cytokines interleukin-6 (IL-6), interleukin-8 (IL-8), and interleukin-1(IL-1)「 IL-1is considered one of the master regulators of the SASP 」 in OIS: chemokines binding to the IL-8 receptor C-X-C motif chemokine receptor 2...
Obese mice exhibited increased levels of inflammatory cytokines in liver and serum, and mice lacking either interleukin-6 (IL-6−/−) or tumor necrosis factor signaling (TNFR1−/−) were protected from the tumor-promoting effects of HFD (Park et al., 2010). Similarly, HFD was also ...
KSHV-encoded viral interleukin-6 (vIL-6) is implicated in the development of KSHV-induced malignancies; however, the mechanisms underlying vIL-6-induced angiogenesis and tumorigenesis remain undefined. Here, we show that vIL-6 promotes angiogenesis, cell proliferation, and invasion by downregulating ...
Interleukin-6 release from peripheral mononuclear cells is associated to disease activity and treatment response in patients with lupus nephritis. Lupus 2009; 18: 1329–1330. Google Scholar Hagiwara E, Gourley MF, Lee S, Klinman DK . Disease severity in patients with systemic lupus erythematosus ...
cells, with a counter-screen against proliferation of ERα-negative MDA-MB231 breast cancer cells, mRNA levels of the proliferative genes GREB1 and MYC, and modulation of tumor necrosis factor alpha (TNF-α)-induced secretion of the inflammatory cytokine, interleukin-6 (IL-6), in MCF-7 ...
During the interaction between KCs and hepatocytes, the cytokines secreted by KCs, such as tumor necrosis factor-α (TNF-α) and interleukin 6 (IL-6), transform hepatocytes from the resting state to the dividing state [2, 62,63,64]. Once the liver is damaged, KCs may be activated by ...
The mechanisms by which chronic hepatitis B is completely resolved through antiviral therapy are unknown, and the contribution of acquired T cell immunity to hepatitis B surface antigen (HBsAg) seroclearance has not been investigated. Therefore, we measu