Western blot analysis of extracts from HeLa cells, cultured in the absence (-) or presence (+) of ethidium bromide, using MT-ATP6 Antibody (upper) and GAPDH (D16H11) XP® Rabbit mAb #5174 (lower). Ethidium bromide-treated cells are depleted of mitochondrial DNA (mtDNA) and mtDNA-encod...
MT-ATP6 Antibody (PA5-37129) in WB Western blot analysis of MT-ATP6 in Lane 1: HeLa whole cell lysate, Lane 2: A549 whole cell lysate, Lane 3: PC12 whole cell lysate. Samples were incubated with MT-ATP6 polyclonal antibody (Product # PA5-37129) at a dilution of 1:500. View ...
中文名称:兔抗MT-ATP6多克隆抗体 英文名称:Anti-MT-ATP6 rabbit polyclonal antibody 别名:mitochondrially encoded ATP synthase 6; ATPase6; MTATP6; ATP6 相关类别:一抗 储存:冷冻(-20℃) 抗原:MT-ATP6 宿主:Rabbit 反应种属:Human 标记物:Unconjugate ...
鼠神经生长因子对语言发育迟缓作用大。鼠神经生长因子是一种由神经元和神经胶质产生的活性多肽,具有促进神经细胞生长和再生的作用。鼠神经生长因子在临床上主要用于治疗神经系统疾病,如脑血管病、颅脑外伤、神经损伤、中枢神经系统感染等。也可以促进神经细胞和突触的生长发育,还可以增加神经元和突触数量。此外,还能增强学...
Gene(s):MT-ATP6 Mutation(s): m.8993T>G, m.8993T>C, m.9185T>C Heteroplasmic Prevalence: Rare Neurogenic muscle weakness,ataxia, andretinitis pigmentosa(NARP) is caused by three mutations inMT-ATP6(m.8993T>C/G, m.9185T>C), all of which also causeMILS(see above). Generally spea...
Mitochondrial complex V (CV) generates cellular energy as adenosine triphosphate (ATP). Mitochondrial disease caused by the m.8993T>G pathogenic variant in the CV subunit gene MT-ATP6 was among the first described human mitochondrial DNA diseases. Due to a lack of clinically available functional ...
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RECOMBINANT THUNNUS OBESUS ATP SYNTHASE SUBUNIT A(MT-ATP6) CBNumber: CB315633117 分子式: 分子量: 0 MOL File: Mol file 化学性质安全信息用途供应商1 RECOMBINANT THUNNUS OBESUS ATP SYNTHASE SUBUNIT A(MT-ATP6)化学性质 安全信息 RECOMBINANT THUNNUS OBESUS ATP SYNTHASE SUBUNIT A(MT-ATP6)性质、用途与...
The most common gene associated with complex V deficiencies, is MT-ATP6 (OMIM *516060), which encodes subunit a of the F0 functional domain [1,2]. This subunit contains the proton pore that releases the proton gradient by conducting protons from the intermembrane space to the matrix. Due ...
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