Lu, "Icariin improves eNOS/NO pathway to prohibit the atherogenesis of apolipoprotein E-null mice," Canadian Journal of Physiology and Pharmacology, vol. 95, no. 6, pp. 625-633, 2017.Xiao, H.B., G.G. Sui and X.Y. Lu, 2016. Icariin improves eNOS/NO-pathway to prohibit the ...
Alexander W., CaulkJose, SolerManu O., PlattRudolph L., Gleason. (2015) Efavirenz treatment causes arterial stiffening in apolipoprotein E-null mice. Journal of Biomechanics /Alexander W.Caulk, JoseSoler, Manu O.Platt, Rudolph L.Gleason. (2015) Efavirenz treatment causes arterial stiffening in ...
Apolipoprotein E (apoE) null mice are a very popular model for studying spontaneous hypercholesterolemia, but only limited data are available for the role of apolipoprotein E in kidney disease. The purpose of this study is to evaluate kidney disease in apolipoprotein E deficient mice. For this ...
Macrophage-specific expression of human apolipoprotein E reduces atherosclerosis in hypercholesterolemic apolipoprotein E-null mice J Clin Invest, 96 (1995), pp. 2170-2179 View in ScopusGoogle Scholar 42 S. Fazio, V.R. Babaev, A.B. Murray, A.H. Hasty, K.J. Carter, L.A. Gleaves, et ...
Fine particulate matter (PM2.5) exposure accelerates atherosclerosis and contains known ovotoxic chemicals. However, effects of exposure to PM2.5 on the finite ovarian follicle pool have hardly been investigated, nor have interactions between ovarian and
Notably, APOE4 itself has been linked to impaired BBB function. In transgenic mice, expression ofAPOE4(but not otherAPOEvariants) leads to breakdown of the BBB; this phenomenon is mediated by the activation of a proinflammatory pathway in pericytes (i.e., perivascular cells) [95]. BBB breakd...
Atherosclerosis in C3H/HeJ Mice Reconstituted With Apolipoprotein E-Null Bone Marrow 来自 NCBI 喜欢 0 阅读量: 55 作者: Shi,W.摘要: Previous studies showed that reconstitution of -susceptible C57BL/6 (B6) female with ()-deficient ((-/-)) bone marrow resulted in markedly increased , despite...
Based on these data, the goal of the present study was to evaluate the protective role of endogenous female sex hormones on hypercholesterolemia-induced renal dysfunction in apoliprotein-E deficient mice, an experimental murine model of hypercholesterolemia and atherosclerosis. Our data demonstrate that...
Macrophage-specific expression of human apolipoprotein E reduces atherosclerosis in hypercholesterolemic apolipoprotein E-null mice J. Clin. Invest., 96 (1995), pp. 2170-2179 View in ScopusGoogle Scholar 41 M. Van Eck, N. Herijgers, K.W. Van Dijk, L.M. Havekes, M.H. Hofker, P.H.E...
Plasma insulin levels predict the development of atherosclerosis when IRS2 deficiency is combined with severe hypercholesterolemia in apolipoprotein E-null mice Front. Biosci., 12 (2007), pp. 2291-2298 View at publisherCrossrefView in ScopusGoogle Scholar 46 L.G. Lyngdorf, S. Gregersen, A. Daug...