Objective To explore the effect of Lctobacillus casei on atherosclerotic plaque formation inapoE-deficient miceand its mechanism in anti-atherogenelsis. 方法将apoE基因缺陷(ApoE-/-)小鼠随机分为4组:对照组(A组)、乳酸杆菌108组(B组)、乳酸杆菌1010组(C组)、乳酸杆菌1012(D组)。
Suppression of proatherogenic leukocyte interactions by MCS-18 -- impact on advanced atheroscle- rosis in ApoE-deficient mice. Atherosclerosis 245, 101-110.Kuehn C,Tauchi M,Stumpf C,et al.Suppression of proatherogenic leukocyte interactions byMCS 18-Impact on advanced atherosclerosis in ApoE ...
Caprylic acid, Inflammation, TLR4, Atherosclerosis, apoE-deficient miceBackgroundAs reported previously by our group, medium-chain triglycerides can ameliorate atherosclerosis. Given that TLR4 is closely related to atherosclerosis, we hypothesized herein that caprylic acid (C8:0) would suppress inflammation...
in Apoe-deficient mice Blockade of IL-17A reduced atherosclerosis5个回答 IL-17A的ApoE基因缺陷小鼠封锁减少动脉粥样硬化2013-05-23 12:21:38 回答:匿名 在Apoe短少老鼠IL-17A封锁减少了动脉粥样硬化 2013-05-23 12:23:18 回答:匿名在IL-17A减少的动脉粥样硬化Apoe短少老鼠封锁 2013-05-23 12:24:58 回答:...
1) apolipoprotein E-deficient mice ApoE基因缺陷小鼠1. OBJECTIVE To investigate the effect of water extractives of Qixue Bingzhi Fang(CWQB) on atherosclerotic labilization plaque pathology and ultrastructure(36 weeks),MMP-1 and TIMP-1 activity in apolipoprotein E-deficient mice(ApoE-mice). 目的...
To clarify the role of this cytokine, we administered thalidomide, a compound known to inhibit TNF-alpha production, to homozygous apolipoprotein E-deficient (apoE-/-) mice in order to examine the effect of thalidomide on the development of early atherosclerotic lesions. Twelve apoE-/- mice were...
(GLP-1) metabolism, we investigated the role of DPP4 in stress-related vascular senescence and atherosclerosis in apolipoprotein E-deficient (ApoE–/–) mice, with a special focus on the pleiotropic effect of adiponectin (APN).Methods: ApoE–/– mice fed a high-fat (HF) diet were randomly ...
Conclusion Salusin-β, but not salusin-α, promotes vascular inflammation in apoE-deficient mice via the I-κBα/NF-κB pathway. These findings provide further insight into the mechanism of salusins in atherosclerosis and potential targets for the prevention and treatment of atherosclerosis. 展开 ...
Xiao Y, Li P, Cheng Y, Zhang Q, Wang F. Effect of α-linolenic acid- modified low molecular weight chondroitin sulfate on atherosclerosis in apoE-deficient mice. Biochim Biophys Acta. 2016;1860:2589-2597.Xiao Y,Li P,Cheng Y,et al.Effect of α linolenic acid modified low molecular ...
Salusin-β Not Salusin-α Promotes Vascular Inflammation in ApoE-Deficient Mice via the I-κBα/NF-κB Pathway 机译:Salusin-β而非Salusin-α会通过I-κBα/NF-κB途径促进ApoE缺陷小鼠的血管炎症 代理获取 本网站仅为用户提供外文OA文献查询和代理获取服务,本网站没有原文。下单后我们将采用程序或人工为您...