(2000) Thromboxane A2 receptors: Where have you gone? Prostagl & Oth Lip Mediat. 60, pp.175-89.Halushka PV (2000) Thromboxane A2 receptors: where have you gone? Prostaglandins Other Lipid Mediat 60:175-189Halushka, P. V. (2000). Thromboxane A(2) receptors: where have you gone?
Clinical studies of patients with mutations in the four major FHM genes have shown that attacks of HM are similar regardless of the gene involved, and that prolonged auras with confusion are possible in all FHM types. The association of HM with epilepsy is present in 7% of overall HM patients...
In addi- tion, as previously described in this review, melatonin is produced by cells and organs of the immune system, which also present melatonin receptors. Although knowledge of the physiological actions of local immune-derived melatonin is limited, there are some pieces o...
On the other hand, active targeting involves modifying the drug or nanocarrier with specific ligands that bind to unique receptors that are overexpressed on target cells. This method, known as ligand-based targeting, enables nanoparticles to bind to specific biomarkers on cancer cells, enhancing drug...
Both, G1202R and S1206Y, are located at the solvent front of the kinase domain and presumably interfere with inhibitor binding due to steric hindrance and conformational changes of the kinase. While the insertion of a threonine residue at 1151 position is speculated to lead to a change in ...