Mitotic centromere-associated kinase (MCAK/Kif2CCellular senescencep53Human primary cellsNot Availabledoi:10.1016/j.bpj.2011.11.2646Ray, SujayQureshi, Mohammad H.Malcolm, DominicCelik, UgurPortman, John J.Balci, HamzaElsevier Inc.Biophysical Journal
DNA processing is not required for ATM-mediated telomere damage response after TRF2 deletion. Nat. Cell Biol. 7, 712–718 (2005). CAS PubMed Google Scholar Sfeir, A., Kabir, S., van Overbeek, M., Celli, G. B. & de Lange, T. Loss of Rap1 induces telomere recombination in the...
1068–1071 (2007).In this publication, the authors demonstrate that ATM and ATR signalling are repressed by TRF2 and POT1, respectively, and that efficient end joining of deprotected telomeres is dependent on DNA damage signalling.
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Hundreds of G4 ligands are reported to bind these sequences with high specificity and affinity, but such ligands can also be engineered to do more than simply associate with G4 in a straightforward hostゞuest fashion. Ligands have been developed that can switch G4 topology, direct the ...
QUADRUPLEX nucleic acidsNUCLEIC acidsThe Cover Feature shows examples of small molecules that unfold G‐quadruplex (G4) nucleic acid structures. Hundreds of ligands bind G4 with high specificity and affinity, an adventure through the universe of these chemotypes discover molecules that can do more ...
This demonstrates the value of mass spectrometry for assessing not only ligand binding, but also ligand-induced rearrangements in the target sequence. This article is part of a Special Issue entitled "G-quadruplex" Guest Editor: Dr. Concetta Giancola and Dr. Daniela Montesarchio. Copyright 漏 ...
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