Suppressed calbindin levels in hippocampal excitatory neurons mediate stress-induced memory loss. Cell Rep. 2017;21:891–900. CAS PubMed Google Scholar Yang XD, Liao XM, Uribe-Marino A, Liu R, Xie XM, Jia J, et al. Stress during a critical postnatal period induces region-specific ...
Figure 1. CD4+ T Cells Play an Essential Role in Stress-Induced Anxiety-like Behavior (A–D) Schematic of experimental design. ES, electronic foot shock; NT, nontreated; i.v., intraperitoneal injection. The treatment with indicated antibodies are only for (C) and (E). (B) Representative...
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miRNA maturation. A similar regulatory pathway is demonstrated for the double-stranded RNA-binding protein DDX1, which also promotes the maturation of a subset of miRNAs (such as the miR-200 family), and the majority of them are induced by DNA damage [52]. Phosphorylation of DDX1 by stress...
CDM, as described in the previous section, has been employed by some researchers to model damage-induced inelastic phenomena in, for example, arterial tissues where the basis of the damage models is a hyperelastic model. Various forms of the SEF were proposed in the literature for soft biologica...
This suggested that these variations are primarily related to induced differences in the balance between supply and demand for water. Hence, studies of {au13}C abundance can disentangle the role of water as such from its effects on mineralization of N and flow of N. 展开 ...
(FSK, 80μmol) induced tau hyperphosphorylation in the rat brain and resulted in significant spatial working memory impairments in Y-maze test, accompanied by synaptic dysfunction (reduced expression of synaptic proteins synaptophysin and postsynaptic density protein 95), and neuronal loss in the ...
b Bodyweight loss induced by repetitive tail-shock stress induction. Mice were weighed on days 1 and 4. Ctrl: control animals, stress: stressed animals. c, d The effect of repetitive tail-shock on immobility in the tail suspension test (TST, c, Ctrl: n = 15, stress: n = ...
Hypoxia (HYPX) induced-overload Ca2+ entry results in increase of mitochondrial oxidative stress, inflammation and apoptosis in several neurons. Ca2+ permeable TRPM2 channel was gated by ADP-ribose (ADPR) and reactive oxygen species (ROS), although its a