在数量性状的GWAS研究里,我们经常会看到一句话:phenotypic variance explained(以下简称PVE)by this QTL is 5%,翻译过来就是这个QTL解释了表型方差的5%。这个数值在很多数量性状(例如,身高、产量)的GWAS结果中都有输出,通常在表头中用R2来表示。 这个指标是什么意思呢?其实就是一个简单的与方差相关的一个指标。现在...
A total of 18 significant KTW-related single-nucleotide polymorphisms (SNPs) were identified using genome-wide association study (GWAS); they were closely linked to 12 candidate genes. In the IBM Syn10 DH population, linkage analysis detected 19 common quantitative trait loci (QTL), five of ...
A total of 18 significant KTW-related single-nucleotide polymorphisms (SNPs) were identified using genome-wide association study (GWAS); they were closely linked to 12 candidate genes. In the IBM Syn10 DH population, linkage analysis detected 19 common quantitative trait loci (QTL), five of ...
In the present study, a marker-trait association analysis was performed on a lemon population employing an association mapping approach. An inter-specific full-sib population composed of 109 accessions was phenotyped through a detached-leaf assays performed in modified Huffaker cells. Those individuals...
associations [5,6]. In response to this, molecular quantitative trait locus (molecular QTL, henceforth “QTL”) analysis has emerged as an important field in human genetics, interrogating the relationship between genetic variants and intermediate, molecular traits and potentially explaining GWAS findings...
根据限制性两阶段多位点全基因组关联分析(restricted two-stage multi-locus genome-wide association analysis,RTM-GWAS)方法,首先构建获得15 546个具有复等位变异的SNPLDB标记,然后使用两阶段多位点模型对百粒重性状进行全基因组关联分析。对检测到的百粒重关联SNPLDB标记位点附近(50 kb范围内)的基因进行分析,根据...
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根据限制性两阶段多位点全基因组关联分析(restricted two-stage multi-locus genome-wide association analysis,RTM-GWAS)方法,首先构建获得15 546个具有复等位变异的SNPLDB标记,然后使用两阶段多位点模型对百粒重性状进行全基因组关联分析。对检测到的百粒重关联SNPLDB标记位点附近(50 kb范围内)的基因进行分析,根据...
QTL detected with linkage (whole average genome interval mapping – WGAIM [DH-QTL], statistical machine learning-SML [DH-QTL], and genome-wide association analysis [GWAS] in Brassica napus germplasm. 来自 figshare.com 喜欢 0 阅读量: 16 作者:...
Genetic susceptibility to colonic inflammation is poorly defined at the gene level. Although Genome Wide Association studies (GWAS) have identified loci in the human genome which confer susceptibility to Inflammatory Bowel Disease (Crohn’s and Ulcerativ