Particularly, we revealed that miR-217 modulates the antibacterial and antiviral immunity through TAK1-mediated NF-魏B and IRF3 signaling pathways. The collective results indicate that miR-217 acts as a negative feedback regulator involved in host antibacterial and antiviral immune responses, which ...
IRF3 抗体IRF3 抗体:偶联ATTO 390IRF3 抗体:偶联ATTO 488IRF3 抗体:偶联ATTO 565IRF3 抗体:偶联ATTO 594IRF3 抗体:偶联ATTO 633IRF3 抗体:偶联ATTO 655IRF3 抗体:偶联ATTO 680IRF3 抗体:偶联ATTO 700IRF3 抗体:偶联碱性磷酸酶IRF3 抗体:偶联APCIRF3 抗体:偶联BiotinIRF3 抗体:偶联FITCIRF3 抗体:偶联HRPIRF3 抗体:...
Inducible MicroRNA-217 Inhibits NF-kappa B- and IRF3-Driven Immune Responses in Lower Vertebrates through Targeting TAK1 Upon recognition of bacterial or viral components by pattern recognition receptors, cells could be activated to produce inflammatory cytokines, type I IFN,... L Zhang,Q Chu,R ...
Reimer T, Brcic M, Schweizer M, Jungi TW. poly (I:C) and LPS induce distinct IRF3 and NF-kappaB signaling during type-I IFN and TNF responses in human macrophages . J Leukoc Biol . 2008; 83 (5) :1249-57. PubMed Abstract Publisher Full Text...
This aggregation initiates intracellular signaling and activates transcription factors, such as nuclear factor-kappa B (NF-κB) and interferon response factor 3 (IRF3), which produce pro-inflammatory cytokines [9,11]. LPS rapidly activates NF-κB and c-Jun N-terminal kinase (JNK) signaling ...
factors, such as nuclear factor-kappa B (NF-κB) and interferon response factor 3 (IRF3), which produce pro-inflammatory cytokines [9,11]. LPS rapidly activates NF-κB and c-Jun N-terminal kinase (JNK) signaling pathways in THP-1 monocytes [12]. These pathways activate various ...