Nature Communications volume 10, Article number: 3452 (2019) Cite this article 20k Accesses 166 Citations 39 Altmetric Metrics details An Author Correction to this article was published on 15 March 2022This article has been updatedAbstract Bacteria have been extensively utilized for bioimaging, ...
Nature Communications volume 10, Article number: 4533 (2019) Cite this article 6732 Accesses 25 Altmetric Metrics details Abstract Multiple myeloma is an incurable, bone marrow-dwelling malignancy that disrupts bone homeostasis causing skeletal damage and pain. Mechanisms underlying myeloma-induced bone ...
DNA binding motifs were retrieved from JASPAR (Fornes et al. 2019), an open-access database of curated, non-redundant binding profiles of DBPs (a.k.a. motifs) stored as position frequency matrices (PFMs). To detect the binding motifs, we used FIMO from the MEME-suite software (https://...
今年 6 月，该团队又在 Nature Communications 上发表了“iPLA2β-mediated lipid detoxification controls p53-driven ferroptosis independent of GPX4”，再次指出 iPLA2β 是在高 ROS 应激条件下 p53 激活诱导的铁死亡的关键调节因子。同时，p53 以不依赖 GPX4 的方式诱导铁死亡。图 7. ALOX12 和 iPLA2β 在调...
Aerial manipulators can manipulate objects while flying, allowing them to perform tasks in dangerous or inaccessible areas. Advanced aerial manipulation systems are often based on rigid-link mechanisms, but the balance between dexterity and payload capac
9月27日,山东师范大学生命科学学院细胞结构与功能研究中心周军教授团队在国际知名学术期刊Nature Communications在线发表了题为Lipid droplets sequester palmitic acid to disrupt endothelial ciliation and exacerbate atherosclerosis in male mice的研究...
今年6 月,该团队又在Nature Communications上发表了“iPLA2β-mediated lipid detoxification controls p53-driven ferroptosis independent of GPX4”,再次指出 iPLA2β 是在高 ROS 应激条件下 p53 激活诱导的铁死亡的关键调节因子。同时,p53 以不依赖 GPX4 的方式诱导铁死亡。
今年6 月,该团队又在 Nature Communications 上发表了“iPLA2β-mediated lipid detoxification controls p53-driven ferroptosis independent of GPX4”,再次指出 iPLA2β 是在高 ROS 应激条件下 p53 激活诱导的铁死亡的关键调节因子。同时,p53 以不依赖 GPX4 的方式诱导铁死亡。
今年6 月,该团队又在 Nature Communications 上发表了“iPLA2β-mediated lipid detoxification controls p53-driven ferroptosis independent of GPX4”,再次指出 iPLA2β 是在高 ROS 应激条件下 p53 激活诱导的铁死亡的关键调节因子。同时,p53 以不依赖 GPX4 的方式诱导铁死亡。
今年6 月,该团队又在Nature Communications上发表了“iPLA2β-mediated lipid detoxification controls p53-driven ferroptosis independent of GPX4” ,再次指出 iPLA2β 是在高 ROS 应激条件下 p53 激活诱导的铁死亡的关键调节因子。同时,p53 以不依赖 GPX4 的方式诱...