Mutation du facteur V Leiden et thrombose : etude de 100 patients symptomatiques en medecine interneWe report upper limb artery thrombosis in two cases of systemic disease: one in a woman with chronic ulcerative colitis and the other revealing systemic sclerosis in a 38 year old woman....
(1998) Détection directe par PCR de la mutation du facteur V Leiden: analyse routinière sur plus de 1 000 échantillons cliniques. Immuno-analyse & Biologie Spécialisée 13 , 362-365 /C Bali, C Bathelier, G Lucotte. (1998) Détection directe par PCR de la mutation du facteur V ...
Livedoid vasculitis is a clinico-pathological entity which may be idiopathic or secondary to various disorders. The aim of this study was to search for a thrombogenic biological abnormality in patients with apparently idiopathic livedoid vasculitis. All the patients with histologically confirmed and ...
Interaction du facteur V Leiden, de la mutation G20210A de la prothrombine et du variant C677T de la MTHFR, dans la maladie thromboembolique veineuse : étude PathrosReport on the Political Science Doctoral Thesis (Paris Dauphine) of Philippe Blanchard: The Media and the Agenda of Nuclear ...
antiphospholipid syndrome / factor V Leiden mutation / resistance to activated protein CRecurrent thrombosis is a common complication of various rheumatic disorders and is part of the definition of antiphospholipid syndrome. We report three cases of recurrent venous thrombosis due not only to ...
Factor V Leiden (FVL) mutation is presently recognized as the most common inherited cause of thrombosis. From a case report the link between varicella associated thrombosis and FVL is discussed. CASE REPORT: An eight year-old boy was admitted on 15th day of a varicella infection for difficulty...
Factor V LeidenThe Leiden mutation of the factor V gene and the subsequent resistance of factor V to inactivation by activated protein C are associated with a procoagulant state, especially in the venous bed. However, its association with arterial thrombotic disease remains unclear. We report four...
In most cases it is due to factorV Leiden mutation by nucleotide G1691A substitution. This recently described thrombophilic defect of activated proteinC resistance has been postulated to be implicated in the pathogenesis of idiopathic intracranial hypertension (IIH). We report a case of factorV ...