Here, we review recent studies describing the molecular mechanisms by which ZIKV induces neural cell death.Zika Virus Biology, Transmission, and Pathologydoi:10.1016/B978-0-12-820268-5.00008-0Trisha R. BarnardQuinn H. AbramSelena M. Sagan
More information:Hao Song et al. Zika virus NS1 structure reveals diversity of electrostatic surfaces among flaviviruses,Nature Structural & Molecular Biology
Molecular evolution emerged as a scientific field in the 1960s as researchers frommolecular biology, evolutionary biology, and population geneticssought to understand recent discoveries on the structure and function of nucleic acids and proteins. This chapter investigates how nucleotide sequences change ove...
According to one recent estimate, dengue virus is responsible for 390 million infections annually, of which 96 million are accompanied by clinical manifestations. Many other arboviruses are also major human pathogens; for example, yellow fever, Japanese encephalitis, West Nile, Zika, and tick-borne ...
Zika virus (ZIKV) is an emerging arbovirus of the genus Flaviviridae, which causes a febrile illness and has spread from across the Pacific to the Americas in a short timeframe. Convincing evidence has implicated the ZIKV to incident cases of neonatal microcephaly and a set of neurodevelopmental...
Zika virus (ZIKV) is an enveloped, icosahedral flavivirus that has structural and functional similarities to other human flavivirus pathogens such as dengue (DENV), West Nile (WNV) and Japanese encephalitis (JEV) viruses. ZIKV infections have been linked
MeSH词表主题词:Gene Transfer Techniques; Genetic Therapy*; Molecular Biology 备注:Title from cover. Also issued online.展开▼ 期刊论文 热门论文 年度选择 2021 Volume 29(1)2021 Jan 6 Volume 29(2)2021 Feb 3 Volume 29(3)2021 Mar 3 2020 ...
Nature Structural & Molecular Biology volume 28, pages 319–325 (2021)Cite this article 35k Accesses 111 Altmetric Metrics details Abstract The COVID-19 pandemic caused by nonstop infections of SARS-CoV-2 has continued to ravage many countries worldwide. Here we report that suramin, a 100-...
secretion of interleukin-1beta did not change, whereas in the presence of the NS3 protein of the hepatitis C virus, it increased by 1.5 times. Thus, NS3 can be considered as one of the factors of NLRP3 inflammasome activation and inflammatory pathogenesis in chronic hepatitis C virus infection...
Biology of Zika virus infection in human skin cells. J. Virol. 2015, 89, 8880–8896. [CrossRef] [PubMed] 65. Bowen, J.R.; Zimmerman, M.G.; Suthar, M.S. Taking the defensive: Immune control of Zika virus infection. Virus Res. 2017. [CrossRef] [PubMed] 66. Sun, X.; Hua, ...