1.mir-493基因启动子分子标记及其引物在秦川牛体尺、肉质鉴定及选种中的应用,所述mir-493基因启动子分子标记的序列如siq id no.1、siq id no.2、siq id no.3或/和siq id no.4所示;其中: 2.根据权利要求1所述的应用,其特征在于,所述引物为: 3.mir-493基因启动子分子标记引物在制备秦川牛体尺、肉质鉴...
在Ⅰ～Ⅱ期PHC患者血清中miR-142-3p、miR-493-5p表达水平显著低于Ⅲ期和Ⅳ期,分期越高其表达水平越低,进一步证实了血清miR-142-3p、miR-493-5p水平变化与PHC患者病情密切相关。通过ROC曲线分析发现,血清miR-142-3p和miR-493-5...
结论miR-493-5p可抑制卵巢癌SKOV3细胞的增殖、迁移和侵袭，其机制可能与靶向调控GIT1以及MEK1/2-ERK1/2通路有关,并可为卵巢癌的诊断、预防和治疗提供新靶点。关键词：miR-493-5p；GIT1；MEK1/2-ERK1/2通路；卵巢癌;侵袭 中图分类号:R737.31文献标志码:A miR-493-5p inhibits the proliferation and ...
oar-miR-493-3p qPCR试剂盒 分类:microRNA qPCR Kits 品牌:BioTNT 货号:oar-miR-493-3p 原价:￥4030 现价:￥3098.38 节省:￥932 规格:1 kits 说明:oar-miR-493-3p 特异性茎环引物试剂盒 ￥615产品参数 ---说明书下载--- microRNAs(miRNAs)是一类长度约19~24nt的非编码单链RNA,通过碱基互补配对的方...
study, we use cell culture and transfection, the tissue specimens, RNA isolation and RT-PCR, Western blot and so on to explore the role of miR-493 in esophageal cancer.Results Overexpression of miR-493 attenuates esophageal cancer cell proliferation, migration, and invasion in vivo and in ...
产品名称 mmu-miR-493 Primersmmu-miR-493 Primers is available 1 time 来自 Abm microrna labs 产品货号 MPM00553 产品价格 现货询价,电话:010-67529703 产品规格 产品品牌 Abm microrna labs 产品概述 mmu-miR-493引物 mmu-miR-493引物可从Abm microrna实验室获得1次 MPM00553 | mmu-miR-493引物大小:...
altered expression levels of FUT4 modified the invasive activities and tumorigenicity of the MCF-7 and MDA-MB-231 cells. Further study demonstrated that miR-493-5p plays a role as a suppressor in breast cancer cell invasion and tumorigenicity. Moreover, the expression levels of miR-493-5p were...
We further found that up-regulation of miR-493 inhibited the proliferation and metastasis of gastric cancer cells,in vitroandin vivo. In addition, miR-493 directly targeted RhoC, which resulted in a marked reduction of the expression of mRNA and protein. This effect, in turn, led to a ...
miR-493-5p suppresses hepatocellular carcinoma cell proliferation through targeting GP73 Biomedicine & Pharmacotherapy Volume 90,June 2017, Pages 744-751 Purchase options CorporateFor R&D professionals working in corporate organizations. Academic and personalFor academic or personal use only. ...
并在SV皮损边缘组织验证了两者的表达减少;采用细胞实验证明了miR-493-3p是通过hnRNPU-COMT轴调节的角质形成细胞多巴胺代谢过程.结论:本研究证明了循环外泌体miR-493-3p可以通过靶向hnRNPU-COMT轴调节角质形成细胞的多巴胺代谢而参与SV发病的具体机制,提供了潜在的SV治疗靶点,表明循环外泌体miR-493-3p有可能成为SV病情...