Edn1fl/fl;Foxg1-Cre Embryos Display Similar Craniofacial Defects with Edn1-/- EmbryosE. GARCIA
The role of sensory organs and the forebrain for the development of the craniofacial shape as revealed by Foxg1-cre mediated microRNA loss. Genesis. 2011Kersigo J, D’Angelo A, Gray BD, Soukup GA, Fritzsch B (2011) The role of sensory organs and the forebrain for the development of ...
volumeisnormal,suggesting thatinteractionsbetweencortexanddorsalthalamuspostnatallyproducethefinaladultthalamic phenotype.IntheFoxg1-crelinemaintainedontheC57BL/6Jbackground,theradialdomainofthe cerebralcortexalsoisdisruptedsubstantially,particularlyinsupragranularlayers.However,neither Foxg1heterozygousmiceoftheFoxg1-...
As the telencephalon of heterozygous Foxg1 mice was reported to be normal, this genotype often has been used as the control in subsequent analyses. Here we describe substantial disruption of forebrain development of heterozygous mice in the Foxg1-cre line, maintained on the C57BL/6J background...
No changes are seen in some pituitary hormone mRNAs or circulating products in Foxg1Cre/+; ARfl/y mice.Laura, O’HaraMichael, CurleyMaria, Tedim FerreiraLyndsey, CruickshanksLaura, MilneLee, B. Smith
figure s2 non cardiac defects fo11owing conditiona1 de1etion of ap 2 using foxg1cre embryos at e15 5 show craniofacia1 a b and thymus c defects abbrethroughtions p pa1ate th thymus sca1e 500 mneuralcrestcells