GenesCell differentiation, genes and cancer edited by T. Kakunaga ... [et al.] (IARC scientific publications, no. 92) International Agency for Research on Cancer, 1988doi:10.1136/jcp.42.11.1231-dNR LemoineInternational Agency For Research Oin CancerJournal of Clinical Pathology...
杨四军博士为著名糖尿病专家、留美八年,多年来一直从事外泌体调控线粒体自稳态在mtROS/NLRP3炎症小体激活级联效应在糖尿病发病中的作用与机制的分子研究。先后发表SCI论文20多篇,其中影响因子10分以上的5篇,发表的SCI文章总影响因子超150。获得国家自然科学基金重点项目子课题、国自然面上项目、教育部留学归国人员...
这是迄今为止最全面的跨癌症分析(cross-cancer analysis),也是癌症基因组图谱(The Cancer Genome Atlas)计划最终的成果。 The UCSC Tumor Map helps researchers visualize the dominant patterns found in the TCGA data, such as the cell of origin, molecular histology, 'stemness' or differentiation status, sp...
题目:Non-coding RNAs and ferroptosis: potential implications for cancer therapy 译名:非编码RNA与铁死亡:对癌症治疗的潜在意义 期刊:Cell Death & Differentiation IF:15.828 发表时间:2022.4.14 通讯作者单位:格拉茨医科大学 DOI号:https:...
Cell death and Differentiation provides an accessible source of up-to-date information for scientists and clinicians. It covers programmed cell death, cell death induced by toxic agents, differentiation and their relation to cell proliferation.
Cell Death and Differentiation: 发表中国乳腺癌研究重要成果 2017年10月13日,英国《自然》旗下《细胞死亡与分化》在线发表中国科技大学、复旦大学附属肿瘤医院、厦门大学、中国科学院、安徽省立医院、安徽医科大学第一附属医院、密歇根大学综合癌症中心、复旦大学上海医学院和生物医学研究院的研究报告,发现通过阻断白细胞...
近日,安徽医科大学郑红团队在Cell Death & Differentiation杂志上在线发表了题为CCKBR+ cancer cells contribute to the intratumor heterogeneity of gastric cancer and confer sensitivity to FOXO inhibition的研究论文,揭示了CCKBR+的胃腺癌...
2022年3月17日,《Cell》发表了一篇preview文章《Mapping the long road to cancer》(绘制通往癌症的漫长道路),该文章表示我们体内的每个细胞都会在一生中积累突变,有时不幸的突变组合会引发癌症。一项新研究通过对血液恶性肿瘤患者的单细胞基因组测序,推断出非常详细的癌前进化时间表:在癌症诊断前的几十年里,往往就...
Gremlins Sabotage the Mechanisms of Cancer Stem Cell Differentiation BMP is highly expressed in glioblastoma and promotes differentiation of cancer stem cells (CSCs). Recently, Yan and colleagues found the explanation to this apparent paradox by showing that the antagonist of BMP, Gremlin1, is secrete...
The p53 tumor-suppressor plays a critical role in the prevention of human cancer. In the absence of cellular stress, the p53 protein is maintained at low steady-state levels and exerts very little, if any, effect on cell fate. However, in response to var