2. In the mammary glands, the milk-secreting epithelial cells, which are activated during lactation. amacrine cell A modified nerve cell in the retina that has dendrites but no axon. See: neuron ameloblast cell Ameloblast. anterior horn cell A somatic motor neuron that has its cell body in...
Moreover, whereas a higher frequency of effector CD4 T cells in both blood and CSF was linked to decreased survival, increased frequencies of activated regulatory T (Treg) cells in the blood was associated with improved survival [29]. While the SOD1G93A mouse model has been widely used in ...
淋巴细胞可分为许多表型与功能均不同的群体,如 T 细胞、B 细胞、NK 细胞、LAK 细胞等,T 细胞和 B 细胞还可进一步分为若干亚群。T 细胞主要参与细胞免疫,表达 CD3 抗原,其中,T 细胞又包括辅助 T 细胞(Th)和抑制 T 细胞(Ts),它们分别表达 CD4 和 CD8...
CD4bright, CD3− low side-scatter cells had the same phenotype as the “plasmacytoid T cell” (Figure 1A-B)31 and the lineage-negative (Lin−), HLADR+, CD123+ CD11c− dendritic cell progenitor (DC2p).27 Ninety percent of the CD123+, Lin−, HLADR+ low side-scatter cells (Fig...
After magnetic-activated cell sorting, cells were stained for TCR-β and CD4 and further purified on a FACSAria II cell sorter (BD Biosciences). Donor CD4+ and CD8+ conventional T cells (Tcons) were prepared from splenocytes and lymph nodes, and enriched with CD4 and CD8 MicroBeads (...
36,37 Our results implied that effector cells of innate immunity, rather than activated T cells, might contribute to the systemic immune activation during HIV infection. The reduced amount and impaired function of regulatory T cells were demonstrated to facilitate immune hyperactivati...
While murine CD4+CD39+ regulatory T cells (Treg) co-express CD73 and hydrolyze exogenous (e) adenosine triphosphate (ATP) to immunosuppressive adenosine (ADO), surface co-expression of CD73 on human circulating CD4+CD39+ Treg is rare. Therefore, the ability of human Treg to produce and ...
Chronic graft-versus-host disease (cGVHD) is an autoimmune-like syndrome mediated by pathogenic CD4+ T and B cells, but the function of extrafollicular and germinal center CD4+ T and B interactions in cGVHD pathogenesis remains largely unknown. Here we s
Despite their key regulatory role and therapeutic potency, the molecular signatures of interactions between T cells and antigen-presenting myeloid cells within the tumor microenvironment remain poorly characterized. Here, we systematically characterize t
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