HAYES, K.C.: On the Pathophysiology of Vitamin A Deficiency, Nutr Rev 29: 3-6, 1971.Hayes KC (1971) On the pathophysiology of vitamin A-deficiency. Nutr Rev 29:3–6Hayes KC. On the pathophysiology of vitamin A deficiency. Nutr Rev. 1971 Jan; 29 (1):3–6....
Pathophysiology of vitamin E deficiency in monkeys1,2 In a comparative study of vitamin E deficiency in Old and New World monkeys, 14 cynomolgus and 12 cebus monkeys were selected at 1 year of age from animals raised in our primate nursery and were fed either a diet containing 8% coconut ...
this (much later) resulted in the addition of vitamins in foodstuffs, the existence of vitamin supplements as preventative measures to beriberi (vitamin B1 deficiency), rickets
Thiamine or vitamin B1 deficiency occurs if the recommended daily intake (RDI) is not maintained. However, deficiency may also occur due to impaired intestinal absorption or high excretion rates, such as in people with alcohol dependency, acquired immunodeficiency syndrome (AIDS), and malnutrition.[6...
Osteoporosis is characterized by low bone mass and microarchitecture deterioration of bone tissue, leading to enhanced bone fragility and consequent increase in fracture risk. Evidence is accumulating for an important role of calcium deficiency as the pr
Evidence is accumulating for an important role of calcium deficiency as the process of aging is associated with disturbed calcium balance. Vitamin D is the principal factor that maintains calcium homeostasis. Increasing evidence indicates that the reason for disturbed calcium balance with age is ...
Vitamin D deficiency is a risk factor for developing multiple sclerosis. The PrevANZ trial was conducted to determine if vitamin D3 supplementation can prevent recurrent disease activity in people with a first demyelinating event. As a sub-study of this trial, we investigated the effect of supplem...
The risk of vitamin B1 deficiency should be prevented in case of large administration of glucose, persistent vomiting (food intolerance syndrome), or diarrhea, by delivering an intravenous dose of 500–1000 mg of vitamin B1. The risk of vitamin B12 deficiency is high, even after the first ...
(VitD) receptor and the VitD-activating enzyme CYP27B1, permitting T cells to both activate and respond to VitD. VitD then initiated the transition from pro-inflammatory interferon-γ+TH1 cells to suppressive interleukin-10+cells. This process was primed by dynamic changes in the epigenetic ...
Although these trials were intended to overcome the limitations related to previous RCTs (that is, too low a dose, the lack of baseline measurement of 25OHD levels and cardiovascular end points as secondary outcomes), they did not set vitamin D deficiency as Spinal Cord 1068 Vitamin D and ...