We hypothesized that in the normal brain under hyperoxic condition, T1 shortening and T2 prolongation occur.Uematsu, HTakahashi, MHatabu, HChen, QChin, CWehrli, S LWehrli, F WAsakura, T
PVL, ventriculomegaly, and thinning of the corpus callosum may produce later reduction in cortical gray matter through disrup- tion of cortical association fibers, it is possible that the lesion represented by prolongation of T2 relaxation time is different and does not affect later brain ...
In the nonsclerotic hippocampus of patients with MTS, T2 relaxation time was prolonged by a mean of 3.3 msec [95% confidence interval (CI), 0.8€"5.9 msec; P= 0.01], patients with l-TLE had prolongation of T2 relaxation time by a mean of 4.3 msec (95% CI, 1.8€"7.1 msec; P= ...
F. Magnetic Resonance Imaging of Skeletal Muscle Prolongation of T1 and T2 Subsequent to Denervation. Investigative radiology 23, 365–369 (1988). Article CAS PubMed Google Scholar Iijima, Y. et al. Differences in fatty degeneration of rotator cuff muscles at different sites, as quantified by ...
Occipital lobe gray matter showed the shortest T1, T2, and T2* relaxation times of all brain regions ( P < .05). CONCLUSIONS T1 shortening in anorexics suggests both dehydration and myelin loss, whereas T2 prolongation points toward myelin loss (myelin water has lower T2), which seems to ...
It is generally accepted that the water content in the brain tissue of immature animals is so great as to be responsible for the prolongation of proton relaxation time, namely, the spin-lattice relaxation time (T1) and the spin-spin relaxation time (T2). In assessing edema in the immature ...
In the nonsclerotic hippocampus of patients with MTS, T2 relaxation time was prolonged by a mean of 3.3 msec [95% confidence interval (CI), 0.8€"5.9 msec; P= 0.01], patients with l-TLE had prolongation of T2 relaxation time by a mean of 4.3 msec (95% CI, 1.8€"7.1 msec; P= ...
It was found that IE water gmT times from both white matter and grey matter tissue decreased by approximately the same amount with refocusing interval prolongation from 10ms to 40 ms. Several mechanisms for this dependence were considered, including water exchange, existence of myelin, non-heme ...