ROS and Mitochondrial Membrane Potential Dependent Modulation of Calcium Signaling in the HeartAmblyopiachildrenERG b-waveNot Availabledoi:10.1016/j.bpj.2012.11.2007Chikando, AristideBoyman, LironKhairallah, RamziWilliams, George S.B.Kettlewell, Sarah...
In this chapter, we describe a relation between mitochondrial membrane potential and the rate of ROS formation. We present different methods applicable for isolated mitochondria or intact cells. We also present experiments demonstrating that a magnitude and a direction (increase or decrease) of a ...
In this chapter, we describe a relation between mitochondrial membrane potential and the rate of ROS formation. We present different methods applicable for isolated mitochondria or intact cells. We also present experiments demonstrating that a magnitude and a direction (increase or decrease) of a ...
Control of mitochondrial membrane potential and ROS formation by reversible phosphorylation of cytochrome c oxidase Phosphorylation of isolated cytochrome c oxidase from bovine kidney and heart, and of the reconstituted heart enzyme, with protein kinase A, cAMP and ATP t... I Lee,E Bender,B Kaden...
also reported increased ROS and mitochondrial membrane potential (see (2) in Fig. 2) in HepG2 cells, which are of course liver derived [53]. Undoubtedly, the intensity of TNT-induced ROS generation is largely defined by its metabolism, as observed in rat liver and kidney samples (high ...
Relation Between Mitochondrial Membrane Potential and ROS Formation. In: Palmeira CM, Moreno AJ, editors. Mitochondrial Bioenergetics: Methods and Protocols ... J Suski,M Lebiedzinska,M Bonora,... 被引量: 33发表: 2018年 [Methods in Molecular Biology] Mitochondrial Bioenergetics Volume 1782 || ...
p53 regulates mitochondrial membrane potential through reactive oxygen species and induces cytochrome c-independent apoptosis blocked by Bcl-2. EMBO J. 18, 6027–6036 (1999). Article CAS PubMed PubMed Central Google Scholar Maxwell, S. A. & Rivera, A. Proline oxidase induces apoptosis in ...
Both effects increased proportionally with the dose of CuE; however, the inhibition of proliferation, arrest of mitosis, production of reactive oxygen species (ROS), and loss of mitochondrial membrane potential (DWm) were found to be dependent on the quantity of CuE used to treat the cancer ...
Mitochondria make ATP via OXPHOS that depends on proton motive force (Δp), which is the sum of the mitochondrial pH gradient (ΔpH) and the mitochondrial membrane potential (ΔΨm). In healthy functional mitochondria ΔΨm is high and ROS generation is positively linked to ΔΨm. So, ...
Lowering pHe from 7.2 to 5.8 reduced cell viability by 70% and was paralleled by a decrease in cytosolic pH (pHc), hyperpolarization of the mitochondrial membrane potential (Δψ), increased levels of hydroethidine-oxidizing ROS and stimulation of protein carbonylation. Co-treatment with the ...