and that it functions as a transmitter.E.S. ViziDepartment of Pharmacology and Department of Anatomy, Semmelweis University of Medicine, Budapest, HungaryJ. HámoriDepartment of Pharmacology and Department of Anatomy, Semmelweis University of Medicine, Budapest, HungaryNeurotransmitters in Invertebrates...
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A., and G. Pilar (1980) The release of acetylcholine from post-ganglionic cell bodies in response to depolarization. J. Physiol. (Lond.) 299: 605-619.Johnson, D.A. & Pilar, G. (1980) The release of acetylcholine from post- ganglionic cell bodies in response to depolarization. J. ...
The actions of various opioids were examined on calcium action potentials in the cell somata of guinea pig myenteric neurones and on the release of acetylcholine at synapses onto these cells. The opioids morphine, normorphine, and [D-Ala2, MePhe4, Met5(O)]enkephalin-ol caused membrane hyperpo...
The acetylcholine responsible for these potentials comes from the same pool of transmitter as that liberated by nerve impulses. The observation that the release is blocked by drugs that prevent the accumulation of acetylcholine into synaptic vesicles indicates that the secretion originates from clusters ...
Presynaptic muscarinic effects on the release of acetylcholine depend on the concentration of Ca2+ and on the degree of depolarizationIt has long been known that certain guanidine compounds exert complex effects on synaptic and neuromuscular ... V Dole?Al,S Tu?Ek - 《Neurochemistry International》 ...
From this end-plate preparation a calcium-dependent release of radioactive transmitter can be measured in the absence of cholinesterase inhibitors. 展开 关键词: Phrenic nerve Whole nerve-muscle preparation End-plate preparation Release of [3H]acetylcholine ...
Neither antagonist blocked the light-evoked release of ACh from the retina. Furthermore, light stimulation did not activate the use-dependent block characteristic of MK-801, indicating that the endogenous transmitter did not open the NMDA channel. These results suggest that NMDA receptors do not ...