Hypertension, beta-adrenergic stimulation and agonists such as angiotensin II (Ang II) activate not only ERK1/2 but also p38 and the Jun N-terminal kinase (JNK), leading to pathological heart remodeling. Despite this progress, the mechanisms that activate fibroblasts to cause fibrosis and those ...
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agent pathogenepathogenicityThe hyperfineinteraction of a ground﹕tate D atom with either a ground﹕tate H or a ground﹕tate D atom is treated. For H + D there are 24 hyperfine states with 12 potential〆nergy curves, and for D + D there are 36 hyperfine states with 22 ...
Posternak T,Pollaczek H.De la protection contre l‘hydrolyse enzymatique exercee par les groupes phosphoryles. Etude de la degradation enzymatique d‘un peptide et d‘un polyose phosphoryles. Helv. Chim. Acta . 1941POSTEMAK T,GRAFL S.De la protection contrel′hydrolyse enzymatique exercée ...