Protein S100-A13doi:10.1007/978-3-319-67199-4_103110S100a13
S100A13 protein S100A13 protein(S100A13 protein) CAS: 化学式: 主页 产品 S100A13 protein 中文名S100A13 protein 英文名S100A13 protein 英文别名S100A13 protein
protein S100-A11 protein S100-A12 protein S100-A13 protein S100-A14 protein S100-A16 Protein S100-A17 protein S100-A2 protein S100-A3 protein S100-A4 protein S100-A5 protein S100-A6 protein S100-A7 protein S100-A7A protein S100-A8 protein S100-A9 Protein S100-C Protein S100-F protein S100...
世联博研公司现货供应ORIGENE TECHNOLOGIES品牌订货号为TP322165的Human S100 calcium binding protein A13 (S100A13), transcript variant 2 from ORIGENE TECHNOLOGIES产品
世联博研公司现货供应ORIGENE TECHNOLOGIES品牌订货号为TP310001的Human S100 calcium binding protein A13 (S100A13), transcript variant 1 from ORIGENE TECHNOLOGIES产品
Recombinant human S100A13 protein重组 人S100A13 蛋白 Recombinant human EEF1A1 proteinRECOMBINANT HUMAN EEF1A1 PROTEIN Recombinant human CLU protein人 CLU 蛋白 Recombinant human CPPED1 protein人 CPPED1 蛋白 Recombinant human FOXO1 proteinRECOMBINANT HUMAN FOXO1 PROTEIN ...
For example, S100A13 overexpression contributed to tumor metastasis and poor survival in patients with early-stage non-small cell lung cancer [16]. Low TMEFF2 expression was associated with larger tumor size and advanced stage and poor differentiation in pancreatic cancer cells [17]. It was ...
S100A13 and acidic fibroblast growth factor (FGF1) are involved in a wide array of important biological processes, such as angiogenesis, cell differentiation, neurogenesis, and tumor growth. Generally, the biological function of FGF1 is to recognize a specific tyrosine kinase on the cell surface ...
recombinantS100A13wasusedheretostimulatehumanendothelialcells.AdditionofextracellularS100A13tothecellsresultedinbothendogenousproteintranslocationandproteinuptakefromtheextracellularspace.Totestspecificityofthiseffect,additionofvariousotherS100proteinswasalsoperformed.Interestingly,translocationofspecificS100proteinswasonly...
Prkd1Crispr-Panc1细胞中s100a4、-a6、-a13和-a16转录本的表达也显著上调。此外,在Prkd1Crispr来源的SEV中,s100a4和s100a16mRNAs显著增加 阻断Prkd1Crispr来源的SEV上的ITG-α6显著减少了肺转移的数量。此外,用从Prkd1Crispr细胞挽救性PRKD1表达的SEV治疗小鼠显著削弱肺转移的形成,同时减少了细胞裂解液及外泌体...