This work was further supported by the Swedish VR contract No. 2017-003 (C. Bierlich), NCN under Grant No. 2018/29/B/ST2/00244 (P. Bozek), EU H2020 MSCA ITN Grant agreement MCnetITN3/722104 (S. Chakraborty and M. Utheim), FAPESP under Grant 17/05685-2 (D. D. Chinellato),...
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Most of the coales- cence approaches evaluate the hadrons yields as a con- volution of the quark thermal (at least at low pT) distri- bution functions at a temperature TH 155 MeV and the hadron wave function [163,164]. A statistical homo- geneous color distribution is assumed in ...
Bottom: Stimulation of egress (left) or egress-to-invasion (right) by supplemented lipids in late IDC PfPP1-DD parasites with partial destabilization (100 nM Shld1), expressed in terms of fold-change relative to no-lipid conditions (mean ± s.e.m., number of experiments indicated ...
20.图1示出了本发明中考虑的enpp1多肽(seq id no:7)。点突变参考seq id no:7鉴定,所述seq id no:7也可以称为“母体化合物”或“构建体#770”。标记方案参考seq id no:7所示的编号方案来鉴定氨基酸编号和残基,然后是已取代seq id no:7中的残基的氨基酸。例如,突变c25n指的是天冬酰胺(asn或n)取代...
TH Page,T Xplore - 《IEEE Transactions on Intelligent Transportation Systems》 被引量: 874发表: 2002年 JSTOR: Functional Ecology, Vol. 8, No. 5 (Oct., 1994), pp. 563-572 2. Daily energy expenditure of 32 female Great Tits (Parus major) tending nestlings 11-12 days old (DEE $_operat...
1(enpp1)的多肽包括seq id no:1的氨基酸残基。 25.在一些实施方案中,所编码的多肽包括融合至胞外核苷酸焦磷酸酶/磷酸二酯酶 ‑ 1(enpp1)的天青杀素信号肽。 26.在一些实施方案中,所编码的包括融合至外核苷酸焦磷酸酶/磷酸二酯酶 ‑ 1(enpp1)的天青杀素信号肽的多肽缺乏聚天冬氨酸结构域或带负电...
Kitali46, O. Kivernyk5, T. Klapdor-Kleingrothaus52, M. Klassen61a, C. Klein32, M.H. Klein106, M. Klein91, U. Klein91, K. Kleinknecht100, P. Klimek121, A. Klimentov29, T. Klingl24, T. Klioutchnikova36, F.F. Klitzner114, P. Kluit120, S. Kluth115, E. Kneringer77, ...
This observation underlines th√e importance of data-taking at the LHC, a pp collider, with s decreased to as close as reasonably possible to 1.96 TeV or 1.8 TeV, the energy range of the p p¯ collisions measured at Tevatron. 3.3 Results for the nuclear slope parameter B(t) Let us ...
3c). In addition, PP1γ-KD cells displayed diminished RIPK1 auto-phosphorylation at S166 and no MLKL phosphorylation at S358, suggesting that PP1γ is required for RIPK1 activation and cell death (Fig. 3d). However, PP1γ has a broad range of substrates and is implicated in various ...