4)P300/SP1复合体调节METTL1的转录活性 为了进一步研究导致METTL1转录上调的分子机制,我们首先通过GENECARD、PROMO和JASPAR网站预测了METTL1的潜在转录因子。三个数据库发现SP1和YY1是共享的(图4A)。鉴于SP1在前列腺癌关键基因转录调控中的重要作...
A GENECARD, PROMO, and JASPAR databases were used to predict the potential transcription factor (TF) of METTL1. B-D Effect of SP1 knockdown on METTL1 mRNA (B-C) and protein (D) levels. E Predicting the combination of SP1 and P300 with the online tool STRING. F Co-immunoprecipitations...
A GENECARD, PROMO, and JASPAR databases were used to predict the potential transcription factor (TF) of METTL1. B-D Effect of SP1 knockdown on METTL1 mRNA (B-C) and protein (D) levels. E Predicting the combination of SP1 and P300 with the online tool STRING. F Co-immunoprecipitations...
The mRNA expression of PGC-1 alpha, a master regulator of mitochondrial gedoi:10.1016/j.cardfail.2007.06.125Yasuaki NakagawaKoichiro KuwaharaGenzo TakemuraMasaharu AkaoMasaki HaradaMichio NakanishiSatoru UsamiHideyuki KinoshitaMasataka FujiwaraKenji UeshimaElsevier Inc.Journal of Cardiac Failure...
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A GENECARD, PROMO, and JASPAR databases were used to predict the po- tential transcription factor (TF) of METTL1. B-D Effect of SP1 knockdown on METTL1 mRNA (B-C) and protein (D) levels. E Predicting the combination of SP1 and P300 with the online tool STRING. F Co-...
Various stresses on the heart, such as myocardial infarction and hemodynamic overload, activate the sympathetic nervous system and the renin-angiotensin system, ultimately reach the nuclei of cardiomyocytes, and change the pattern of gene expression associated with cardiac hypertrophy. Although present ...
The objective of this study is to review part of the literature and find evidence for and against the characteristics observed in the P300 and the possible part played by the DRD2 gene in the aetiology of alcoholism and the relationship between them. At the same time we consider the most ...
Aims: We investigated the effect of hypoxemia alone under magnesium exposure on fetal cardiovascular changes in chronically catheterized goat fetuses. Stdoi:10.1016/j.cardfail.2005.08.212Tatsuya MorimotoKoh OnoTeruhisa KawamuraToru KitaKoji HasegawaElsevier Inc.Journal of Cardiac Failure...
In primary neonatal rat cardiac myocytes, stimulation with either ALD or phenylephrine (PE) was sufficient to increase the cell size and to activate hypertrophy-associated gene transcription. Co-stimulation of PE with ALD further augmented these hypertrophic responses. In addition, PE induced expression...