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The non-canonical NF-κB pathway is tightly controlled by ubiquitin-dependent degradation of NIK mediated by an E3 ubiquitin ligase complex composed of cIAP family members, TNFR-associated factor 2 (TRAF2) and TRAF3; activation of non-canonical NF-κB involves signal-induced disruption of the cIA...
non-canonical NF-kB signaling pathway. Cell Res. 21, 71-85. Trosky, J.E., Li, Y., Mukherjee, S., Keitany, G., Ball, H., and Orth, K. (2007). VopA inhibits ATP binding by acetylating the catalytic loop of MAPK kinases. J. Biol. Chem. 282, 34299-34305....
Anderson Cancer Center, The University of Texas Graduate School of Biomedical Sciences at Houston, 7455 Fannin Street, Box 902, Houston, TX 77030, USA The non-canonical NF-κB pathway is an important arm of NF-κB signaling that predominantly targets activation of the p52/RelB NF-κB complex...
The non-canonical NF-κB pathway is crucial for the immune system. A critical event in activation of the non-canonical pathway is the attenuation of NF-κB-inducing kinase (NIK) degradation, which is promoted by continuous polyubiquitination of NIK catalyzed by the NIK ubiquitin-ligase complex co...
We wanted to determine if NRAGE was also required for the activation of NF-κB through the non-canonical BMP pathway. Overexpression of full length NRAGE in 293HEK cells resulted in the constitutive phosphorylation of IKK-α and IKK-β (Figure 1A) while disruption of NRAGE expression through ...
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Two distinct nuclear factor κB (NFκB) signaling pathways have been described; the canonical pathway that mediates inflammatory responses, and the non-canonical pathway that is involved in immune cell differentiation and maturation and secondary lymphoid organogenesis. The former is dependent on the I...
ARTICLE DOI: 10.1038/s41467-018-03907-4 OPEN Non-canonical activation of DAPK2 by AMPK constitutes a new pathway linking metabolic stress to autophagy Ruth Shiloh1, Yuval Gilad1, Yaara Ber1, Miriam Eisenstein2, Dina Aweida3, Shani Bialik1, Shenhav Cohen3 & Adi Kimchi1 Autophagy is an ...
We now show that TWEAK also activates the non-canonical NFκB pathway in podocytes, promoting the expression of the chemokine CCL21 that, as Fn14, is upregulated in vivo in the course of nephrotoxic podocyte injury. NFκB activates the transcription of different genes with specificity...