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(1998). Stable overex pression of the consitutive form of heat shock protein 70 confers oxidative protection. JMCCC 30:599–608. CAS Google Scholar Miller, M.J. (2001). Preconditioning for cardioprotection against ischemia reperfusion injury: the roles of nitric oxide, reactive oxygen species,...
G3-17-NF-Volcanoes Strategies ✓Makingpredictions✓Questioning✓Summarizing HaveyouseenthisonTV?Thisisavolcano.WarmUp WarmUp Therearealotofvolcanoesintheworld.crust eruption AcademicWords lava Thecrustismadeofbigpiecesoflandcalledplates.Thisiscalledaneruption.Whenthegodswereangry,peoplebelievedtheywould...
In particular, we asked whether a dominant-negative form of IkB kinase 2 (dnIKK2)25, a kinase that is an upstream activator of NF-κB, interferes with the expression of miR-503. In cultured ECs, Ad.dnIKK2 reduced the pri-miR-503 and mature miR-503 expression, which were previously ...
In fact, a number of studies have shown that the efficacy of therapeutic drugs could be potentiated by met- formin; some studies have even attempted to elucidate the underlying mechanisms. In accordance with our results, Kim et al. revealed that metformin could activate AMPK and suppresses MDR...
and 2R01DK61707 (to G. N.). This work was also supported by the Crohn's and Colitis Foundation of America and the Eli and Edythe L. Broad Foundation Medical Research Program for Inflammatory Bowel Disease. This article contains supplemental Figs. S1–S11 and Tables S1–S8. 1 Both ...
NF-κB contains the subunits p50/p65, which form a heterodimer in the cell [40]. Under normal physiological conditions, IκB and NF-κB interact so that NF-κB is present in an inactive state [41]. External stimuli induce the ubiquitination of IκB-α in cells, thereby causing the ...
The activated form of NF-κB is a survival signal for tumor cells because the activation of NF-κB leads to expression of several antiapoptotic genes. We examined whether XN can modulate the expression of antiapoptotic genes, which are under the control of NF-κB, such as Bcl-xL, cIAP-1,...
In line with their high functional domain homology, Tax-1 and Tax-2 show interesting similarities in their ability to form complexes with cytoplasmic factors. We demonstrated that both Tax-1 and Tax-2 form complexes with NF-κB pathway components NEMO and p65/RelA, the scaffold protein TAB2,...
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