60 、He'd been rescued by a Blackfoot Indian,had lain unconscious for six days in a Montana hospital's intensive care unit,and ended up with neurological damage and a blind right eye.───在蒙大拿一家医院的特别护理室昏迷了六天,最终还是留下神经损伤和右眼失明的后遗症。 61 、The book will...
The massachusetts general hospital guide to depression. Cham: Springer Nature Switzerland AG; 2019. Book Google Scholar Lampl Y, Zivin JA, Fisher M, Lew R, Welin L, Dahlof B, et al. Infrared laser therapy for ischemic stroke: a new treatment strategy: results of the NeuroThera ...
Department of Neurosurgery, Cooper University Hospital, 1 Cooper Plaza, Camden, NJ, 08103, USA Navid Redjal Department of Neurosurgery, Capital Health Institute for Neurosciences, Pennington, NJ, USA Navid Redjal Department of Neurosurgery, Inova Neuroscience and Spine Institute, Falls Church, VA, US...
The immunomodulatory properties of MSC are key elements for their application to regenerative medicine because the host immune response to tissue damage can result in neural cell death. Cell-to-cell contact and efferocytosis of MSC are known to contribute to immunosuppression [34]. Numerous molecules ...
(pCO2< 25 mmHg) in the prehospital environment had higher mortality, and that use of quantitative capnography by paramedics significantly decreased the incidence of hyperventilation [125]. A similar study in patients with major trauma showed a much higher incidence of “normocapnea” on hospital ...
Detection rate of psychogenic cough in patients with chronic cough in Chinese hospital: a meta-analysis. Int J Clin Exp Med. 2016;9:504-514. [Cited in This Article: 1] 12. Vertigan AE. Somatic cough syndrome or psychogenic cough-what is the difference? J Thorac Dis. 2017;9:831-...
[102]. If infected, phagocytes capture near odour and taste cells, the virus causes oxidative stress damage to the cells, leading to odour and taste disorders. If the rupture is near the meningeal space, the virus produces oxidative stress damage to the cerebral cortex and induces brain ...
CLN2 neuronal ceroid lipofuscinosis is a hereditary lysosomal storage disease with primarily neurological signs that results from mutations in TPP1, which encodes the lysosomal enzyme tripeptidyl peptidase-1 (TPP1). Studies using a canine model for this
Department of Anesthesia, Surgical Intensive Care, Prehospital Emergency Medicine and Pain Therapy, University Hospital Basel, University of Basel, Spitalstrasse 21, CH-4031, Basel, Switzerland Pascal A. Gerster, Anna Klesse, Julia Chang, Joachim M. Erb & Nicolai Goettel Department of Clinical Resear...
Author information Authors and Affiliations PICU/NICU, University Children's Hospital, Inselspital Bern, CH-3010, Bern, Switzerland J. Pfenninger Rights and permissions Reprints and permissions About this article Cite this article Pfenninger, J. Neurological intensive care in children. Intensive Care Med...