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Ohe THeart rhythm: the official journal of the Heart Rhythm SocietyOhe T. Radiofrequency ablation for ventricular tachycardia in pa- tients with cardiac sarcoidosis: is it worth trying? Heart Rhythm. 2009;6(2):196-7.
patients in sinus rhythm: the role of atrioventricular junction ablation. To assess the clinical benefit of cardiac resynchronization therapy (CRT) in patients with atrial fibrillation (AF) compared with patients in sinus rhythm ... António,M,Ferreira,... - Europace : European pacing, arrhythmias...
A decline in T regulatory cells (Treg) was observed in heart failure patients (HFP) when compared to healthy individuals (HG 108050 versus HFP-T0 069040, P=0.0022), and this reduction remained after cardiac resynchronization therapy (CRT) (HFP-T6 061029, P=0.0003). At time zero (T0), ...
A cardiac arrhythmia ablation catheter that has a highly flexible tubular distal segment particularly adapted for navigating and exploring a ventridular cardiac chamber forming a lumen through its length which connects to a distal electr... BOAZ AVITALL ...
In zebrafish embryos treated with 3-PCA at a concentration of 5 mg/L, the results showed abnormal cardiac development and a decrease in heart function. Embryos treated with 3-PCA exhibited a substantial decrease in cacna1c expression, the gene responsible for a voltage-dependent calcium channel....
et al. AEO-7 surfactant is “super toxic” and induces severe cardiac, liver and locomotion damage in zebrafish embryos. Environ Sci Eur 32, 149 (2020). https://doi.org/10.1186/s12302-020-00429-z Download citation Received16 September 2020 Accepted30 October 2020 Published11 November 2020 ...
In the last decade, scEmc10was first shown to be upregulated by high glucose in pancreatic β-cells14. This secreted isoform was later shown to be highly expressed in high-grade gliomas15,16, and to promote cardiac tissue repair after myocardial infarction19. The membrane bound isoform was ...
And Scd1 deficiency is cardioprotective against HFD-induced cardiac dysfunction as well as leptin-deficient mice by stimulation of glucose oxidation [150]. Therefore, SCD1 inhibition provides metabolic benefits in liver, adipose tissue, skeletal muscle and heart, which justifying it as the hub gene ...