The integration of the exogenous copy of rop55 at the uprt locus was verified with primers ML3187 and ML2081 and ML2384 and ML3190. Primer sequences are in Supplementary Table 1. Plaque assay Cells were seeded on 24-well plates and grown to 100% confluency and infected with 7000 tachy...
Certainty in results is a combination of high-resolution, quantitative accuracy and consistent mass accuracy. Incorporating a maintenance-free secondary ion source to deliver a regulated number of calibrant ions into ...
Programmed cell death is a critical aspect of the innate immune response and controls infections by killing and clearing infected cells, thereby depriving intracellular pathogens of their replicative niche9. Cell death pathways can either induce cell lysis and inflammation, as observed in both pyroptosis...
2081.Sum-of-k-Mirror-Numbers (H-) 795.Number-of-Subarrays-with-Bounded-Maximum (M+) 1625.Lexicographically-Smallest-String-After-Applying-Operations (H-) 1714.Sum-Of-Special-Evenly-Spaced-Elements-In-Array (H) 1737.Change-Minimum-Characters-to-Satisfy-One-of-Three-Conditions (M+) 2013.Dete...
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JP2002081528A2002-03-22 JP2000074048A2000-03-07 JPH11201153A1999-07-27 AT289563B1971-04-26 JPH09292008A1997-11-11 JPH08219161A1996-08-27 Attorney, Agent or Firm: Witte, Weller & Partner (Stuttgart) Claims: 1. Wälzlagerkäfig (10) für ein Wälzlager (30), mit einem zylinderring...
208, 2069–2081 (2011). 9. Walker, J. A. et al. Bcl11b is essential for group 2 innate lymphoid cell development. J. Exp. Med. 212, 875–882 (2015). 10. Yu, Y. et al. The transcription factor Bcl11b is specifically expressed in group 2 innate lymphoid cells and is essential ...
Master transcription factors reprogram cell fate in multicellular eukaryotes. Pioneer transcription factors have prominent roles in this process because of their ability to contact their cognate binding motifs in closed chromatin. Reprogramming is pervas
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NOTCH1 alterations have been associated with chronic lymphocytic leukemia (CLL), but the molecular mechanisms underlying NOTCH1 activation in CLL cells are not completely understood. Here, we show that GSK3β downregulates the constitutive levels of the