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Each model included all three time-dependent insulin exposure variables and was adjusted for time-dependent age and duration of insulin-treated diabetes, sex (not in the sex-stratified analysis), baseline calendar time, use of non-insulin antidiabetic drugs (NIADs), other co-medication (when ...
In this context, we have investigated the possibility of risk factor-matched age-dependent differences in prevalence of LHs as the primary endpoint of our study, and the localization of LHs and associated factors as secondary endpoints. Methods This study is a sub-analysis of data collected in ...
decre.~scdfilucosc o h i d a l ~ oa~pipeared only afler #.Iy o f obesily, nlld wo~serlvci v i l l l ubeblly d u ~ n t ) o >(1l-=0.7?, p<.OOOi) We h y p o l i l c s i ~ cIlia1 i~lcrc,~scdl > p i i Iohid,lllcln ircini; one d IcdcI l o ...
decre.~scdfilucosc o h i d a l ~ oa~pipeared only afler #.Iy o f obesily, nlld wo~serlvci v i l l l ubeblly d u ~ n t ) o >(1l-=0.7?, p<.OOOi) We h y p o l i l c s i ~ cIlia1 i~lcrc,~scdl > p i i Iohid,lllcln ircini; one d IcdcI l o ...
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International Journal of Molecular Sciences Article Insulin-Induced Recurrent Hypoglycemia Up-Regulates Glucose Metabolism in the Brain Cortex of Chemically Induced Diabetic Rats Susana Cardoso 1,2,3 and Paula I. Moreira 1,3,4,* 1 CNC—Center for Neuroscience and Cell Biology, University of Coimbra...
11.2.2. .InInssuulilninPProrodduuctcitoionnaannddDDeleilvivereyrytotoTTisissusuesesTThahtatTaTkaekUe Up pInIsnusluinlin InInssuulilniniisspprroodduucceedd by pancreaticc ββcceellslsaannddsescercerteetdedinitnotothtehpeoprotarltavleivne,inw,hwichhicdhe- dleivlievresrsthtehenenwewlylyprpor...
antioxidants Article Low-Dose Dioxin Reduced Glucose Uptake in C2C12 Myocytes: The Role of Mitochondrial Oxidative Stress and Insulin-Dependent Calcium Mobilization Suyeol Im 1, Sora Kang 2, Ji Hwan Kim 1 , Seung Jun Oh 1 and Youngmi Kim Pak 1,2,3,* 1 Department of Biomedical Sciences, ...
In pancreatic cancer PANC-1 cells, crosstalk between GPCRs and IR receptor-signaling pathways has revealed that IR-dependent potentiation of mitogenic agonist, neurotensin, is related to the mammalian target of rapamycin complex 1 (mTORC1) pathway [69]. Following IR activation, the subsequent ...