-15a-5p-IL-6/STAT3/CX3CL1的模型可以调节内皮细胞的细胞增殖迁移过程从而影响动脉粥样硬化的发展.结论:血管损伤的炎症作用贯穿了AS发生,发展的过程,趋化因子与动脉粥样硬化之间的关系受到越来越多的关注,CX3CL1在动脉粥样硬化病理状态下表达增高.本实验证明了IL-6/STAT3通路的激活促进了CX3CL1的表达,而miR-15a...
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