Based on this analysis, we further explored and compared the mechanism of action of IL (interleukin)-1 beta versus TNF-alpha (tumor necrosis factor-alpha) in abdominal aortic aneurysm formation.Batra, RishiSuh, Melissa K.Carson, Jeffrey S.Dale, Matthew A.Meisinger, Trevor M.Fitzgerald, Matthew...
白细胞介素-1B(IL-1B)是促炎细胞因子和急性期反应诱导剂。在微生物诱导后,IL-1B主要由单核细胞,巨噬细胞和树突细胞产生。肿瘤坏死因子-a(TNF-a)由许多不同细胞类型产生,例如 单核细胞,巨噬细胞,T细胞和B细胞。在TNF-a的许多作用中,有针对细菌感染,细胞生长调节,免疫系统调节和参与败血症性休克的保护。
TNF-alpha and IL-1 beta upregulate nitric oxide-dependent ciliary motility in bovine airway epithelium. Am J Physiol 1995;268:L911-L917.Jain B, Rubinstein I, Robbins RA, Sisson JH (1995) TNF-alpha and IL-1 beta upregulate nitric oxide-dependent ciliary motility in bovine airway epithelium....
TNF-alpha and IL-1 beta are not essential to the inflammatory response in LPS-induced airway disease. Benda, E, Pruessner, JA, Schwartz, DA (2001) TNF-alpha and IL-1 beta are not essential to the inflammatory response in LPS-induced airway disease. ... Moreland JGFuhrman RMWohlford ...
TNF-alpha and IL-1 beta are not essential to the inflammatory response in LPS-induced airway disease. Benda, E, Pruessner, JA, Schwartz, DA (2001) TNF-alpha and IL-1 beta are not essential to the inflammatory response in LPS-induced airway disease. ... Moreland JGFuhrman RMWohlford Lenan...
Altered production of PGE2, IL-1β and TNF-α by peripheral blood monocytes from HIV-positive individuals at early stages of HIV infection N Longo,JM Zabay,JM Sempere,... - 《Journal of Acquired Immune Deficiency Syndromes》 被引量: 0发表: 1993年 Reversal of tumor-induced immunosuppression:...
Only TNF-alpha and IL-6 levels were significantly higher in sera of active pulmonary TB patients, 92.3 +/- 28.7 vs. 3.5 +/- 1.2; 15.2 +/- 5.4 vs. 2.1 +/- 2.1, respectively. Northern blot analysis revealed increased gene expression of these alveolar macrophage cytokines in patients with ...
Since proinflammatory cytokines mediate many of the acute responses in critical illness, we evaluated the effects of IL-1 beta and TNF-alpha on growth hormone receptor-(GHR-)mRNA in cultured rat hepatocytes. Diminished GHR-mRNA concentrations in response to cytokine stimulation indicate that low IGF...
《Betatrophin对TNFalpha或IL-1beta诱导NF-kappaB活化的负调控机制研究》是依托华中科技大学，由张煜担任项目负责人的青年科学基金项目。中文摘要 非可控炎症是糖尿病、癌症等多种重大疾病的主要诱因，调控紊乱是造成炎症处于非可控状态的重要原因。NF-kB能诱导一系列炎症相关蛋白表达，在炎症反应调控中发...