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Acute myeloid leukemia (AML) is caused by altered maturation and differentiation of myeloid blasts, as well as transcriptional/epigenetic alterations, all leading to excessive proliferation of malignant blood cells in the bone marrow. Tumor heterogeneity due to the acquisition of new somatic alterations...
Acute myeloid leukemia (AML) is the most common form of acute leukemia in adults, with a dismal 5-year survival rate of only 24%. This prognosis is even worse in refractory or relapsed (R/R) patients, where the 5-year survival rate falls below 10% [1]. Given these bleak statistics,...
Mutations in FMS-related receptor tyrosine kinase 3 (FLT3) are among the most common alterations in acute myeloid leukemia (AML), present in ≈30% of newly diagnosed AML cases. Internal tandem duplications (ITD) in FLT3 (FLT3-ITD) occur in ≈25% of newly
Furthermore, FTO suppresses miR-181b-3p, leading to increased expression of the cancer-promoting gene ARL5B. This, in turn, promotes the movement and invasion of breast cancer cells [138]. In acute myeloid leukemia (AML), ALKBH5 stimulates the growth of AML cells by improving the stability...
CD47 is highly expressed in AML leukemic stem cells and AML cells. Interestingly, CD47 mRNA had lower expression in cases harboring t (8;21), a favorable risk translocation, whereas higher expression is strongly correlated with FLT3-ITD mutations that confer worst survival in AML with normal cyto...