FAM126A 基因家族蛋白 PI4KA lipid kinase complex 官方名通用名 PI4KAPI4KA protein, recombinant FAM126A 蛋白分子量&等电点 参数根据以下信息计算得出 FT CHAIN 1-521 Hyccin. 分子量 (Da) 57625.43 理论PI值 8.45 批量订购FAM126A重组蛋白
FAM126A Antibody (PA5-34761) in WB Western Blot using FAM126A Polyclonal Antibody (Product # PA5-34761). Sample (50 µg of whole cell lysate). Lane A: mouse brain . 7.5% SDS PAGE. FAM126A Polyclonal Antibody (Product # PA5-34761) diluted at 1:3,000. View more 产品...
Target NameFAM126A Accession No.Swiss-Prot:Q9BYI3 Gene ID:84668 UniprotQ9BYI3 Gene ID84668; Concentration1mg/ml FormulationSupplied in 0.1M Tris-buffered saline with 10% Glycerol (pH7.0). 0.01% Thimerosal was added as a preservative.
26243-1-AP targets FAM126A in WB, ELISA applications and shows reactivity with human samples. Tested Applications WB, ELISA Application Description Cited Applications WB Tested Reactivity human Cited Reactivity human Immunogen FAM126A fusion protein Ag24004 种属同源性预测 Host / Isotype Rabbit /...
FAM126A抗体 英文名字 FAM126A antibody 供应商 Fitzgerald 产品货号 70R-10016 产品报价 ￥询价/50ug 产品说明书 点击查看 购买方式 银行转账、电汇、支票、现金,在线支付宝及网银支付,或直接与我们电话联系400-6800-868。 产品新闻 背景资料 Affinity purified rabbit polyclonal FAM126A antibody,FAM126A抗体,...
FAM126A OVEREXPRESSION LYSATE (NATIVE) 中文名: 中文别名: CBNumber: CB34738771 分子式: 分子量: 0 MOL File: Mol file 化学性质安全信息用途供应商1 FAM126A OVEREXPRESSION LYSATE (NATIVE)化学性质 安全信息 FAM126A OVEREXPRESSION LYSATE (NATIVE)性质、用途与生产工艺 ...
Fassad, M.R.Biochemical & Biophysical Research CommunicationsTraverso M, et al. Novel FAM126A mutations in hypomyelination and congenital cataract disease. Biochem Biophys Res Commun. 2013; 439 :369–372.Traverso, M. et al. Novel FAM126A mutations in hypomyelination and congenital cataract ...
Product Name: FAM126A OVEREXPRESSION LYSATE (NATIVE) Synonyms: FAM126A OVEREXPRESSION LYSATE (NATIVE) CAS: MF: MW: 0 EINECS: Mol File: Mol FileInformation Error Report Your Email: Browse by Nationality FAM126A OVEREXPRESSION LYSATE (NATIVE) Suppliers China suppliersJapan...
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Baskin et al. report that FAM126A, which is mutated in hypomyelination and congenital cataract, regulates PI4KIII, the kinase generating PtdIns(4)P. Loss of FAM126A in patients affects PI4KIIIα complex assembly and PtdIns(4)P synthesis at the