norepinephrinecocaineCocaine has long been assumed to raise blood pressure (BP) by blocking norepinephrine (NE) reuptake in sympathetic nerve terminals, resulting in alpha-adrenergic vasoconstriction in the peripheral circulation. However, this assumption has not been tested directly in humans, and our ...
increase in blood pressure d. a and b are correct c. a and c are correct Primary mechanism by which norepinephrine acutely increases BP: A) Increases intravascular volume B) Increases heart rate C) Vasoconstriction at precapillary resistance muscles and veins D) Increases angiotensin II plasma ...
The relationship between plasma levels of norepinephrine (NE) and sympathetic neural activity is discussed with special reference to human primary hyperten... B Folkow,GFD Bona,P Hjemdahl,... - 《Hypertension》 被引量: 512发表: 1983年 Cardiovascular Reactivity in the Laboratory and the Role of...
(n = 32, 46%), sedation (n = 32, 46%), intravenous L-carnitine (n = 19, 28%, administered on ICU admission, once the diagnosis of VPA poisoning was established), norepinephrine (n = 13, 19%), blood transfusion (n = 2, 3%) and hemodialysis (n = 2...
Weber M, Talmon S, Schulze I, Boeddinghaus C, Gross G, Schoemaker H et al (2009) Running wheel activity is sensitive to acute treatment with selective inhibitors for either serotonin or norepinephrine reuptake. Psychopharmacology 203(4):753–762 Article PubMed CAS Google Scholar Wen PH, Sha...
Treatments of mood disorders generally aim to increase the availability of monoamines (norepinephrine, serotonin and dopamine) in the brain. To determine whether the effects of EC are mediated through monoaminergic mechanisms, we performed immunoblotting experiments with homogenized hippocampal and cortical...
Which of the following choices would cause a decrease in cardiac output? a. Application of norepinephrine. b. Use of epinephrine. c. Application of acetylcholine. d. Increase in the end-diastolic volume.An increase in blood vo...
BAT is activated by the sympathetic nervous system (SNS) releasing norepinephrine (NE) that binds to β3-adrenergic receptors (ADRB3) on brown adipocytes, leading to an increase in intracellular cyclic AMP (cAMP) (Zhao et al., 1994). This rise in cAMP activates, for instance, the ...
The pattern of expression we observed for GHR, ADRB2, HSL, and NR3C1 suggested that dietary OVE did not alter sensitivity of SAT to lipolytic stimulation postpartum (e.g., via norepinephrine or cortisol). Growth hormone action on AT antagonizes insulin by amplifying the lipolytic response to...
The onset of UCP gene transcription,which may be considered as the key point of brown fat differentiationin vivo, begins between days 18 and 19 of fetal life in the rat, and is followed by an abrupt increase in UCP mRNA levels (16, 17).BAT S'D-I1 shows peak values on day 20 of ...