SUMMARY The objective of this study was to examine the effects of colcemid treatment during oocyte in vitro maturation (IVM) and embryonic development after parthenogenetic activation (PA) and somatic-cell nucleus transfer (SCNT) in pigs. Immature oocytes were treated with colcemid from 0 to 22, ...
Effect of Colcemid treatment of myeloma (X63-Ag8-6.5.3.) prior to fusion with mouse spleen cell was studied in terms of hybridoma formation. Spleen cells from BALB c mice immunized with various soluble antigens were fused with the myeloma cells by using polyethylene glycol solution. Colcemid ...
Colcemid-treatment of heifer oocytes enhances nuclear transfer embryonic development, establishment of pregnancy and development to term. Mol Reprod Dev, v.76, p.620-628, 2009.Li, G-P., White, K.L., Aston, K.I., Bunch, T.D., Hicks, B., Liu, Y., and Sessions, B.R. (2009). ...
, & Isaacson, R. L. Deficient brain development following Colcemid treatment in postnatal rats. Brain Research , 1977, 132 , 380–385.Petit, T. L. and Isaacson, R. L. (1977) Deficient brain development following colcemid treatment in postnatal rats. Brain Res . 132 , 380–385....
Colcemid treatment severely inhibits the progression of cells in mitosis and causes enrichment of cells into the M phase of the cell cycle. This blockage in M phase was reversible at the drug concentration used, and cells resumed cycle traverse following colcemid removal without any delay. The ...
Clones of CHO cells stably resistant to colcemid have been isolated in the presence of the nonionic detergent Tween 80 after mutagen treatment. Successive ... V Ling,JE Aubin,A Chase,... - 《Cell》 被引量: 132发表: 1979年 Secretion of macrophage neutral proteinase is enhanced by colchicine...
The induction of mouse epidermal ornithine decarboxylase, 1 of the earliest and largest phenotypic changes following treatment of mouse skin with the tumor... TG O'Brien,RC Simsiman,RK Boutwell - 《Cancer Research》 被引量: 589发表: 1976年 Taxol resistant mutants of Chinese hamster ovary cells...
The combination treatment also showed inactivation of non-canonical 尾-catenin signaling suggesting inhibition of cancer growth. In addition, the combination treatment has significantly affected the distribution and functions of bubR1 downstream mitotic kinases such as aurks and plk1 indicating the ...
Roscovitine and colcemid treatment to regulate the MPF activity in MII oocytes.KyeoungHwa, KimEunYoung, KimYuna, KimEunju, KimHyunSeo, LeeSookYoung, YoonKyungAh, Lee
When floating D98/AH 2 cells and normal human fibroblasts (Detroit 550) harvested after 20 hr Colcemid treatment were incubated in Colcemid-free medium, a majority attached to and spread on the substrate, and approximately 50–70% were micronucleate. The failure of some human cells to micro...