Cell Death and Disease is an online journal in the field of translational cell death. It seeks to promote diverse and integrated areas of Experimental and Internal Medicine with its specialties, including Cancer, Immunity and Neuroscience.
Cell death articles withinCell Death & Disease Featured Article 26 January 2025|Open Access Synaptotagmin-1 attenuates myocardial programmed necrosis and ischemia/reperfusion injury through the mitochondrial pathway Teng Sun ,Jialei Li &Jimin Cao
His recent work is focusing on attempts to increase the sensitivity of lung cancer cells to treatment using regulation of cross-talk between various cell death modalities. He was an organizer of three Nobel Conferences on The Cell Cycle and Cell Death in Disease and several other meetings; ...
Besides being part of the Editorial Board of Cell Death and Disease, Lorenzo operates as Editor-in-Chief for OncoImmunology and Molecular and Cellular Oncology. He is particularly fascinated by several aspects of mitochondrial cell death, apoptosis, oncometabolism and tumor immunology. KEYWORDS: cell ...
Tipping the balance of cell death: alternative splicing as a source of MCL-1S in cancer Mariusz L. Hartman Article 18 December 2024|Open Access Tumor- and host-derived heparanase-2 (Hpa2) attenuates tumorigenicity: role of Hpa2 in macrophage polarization and BRD7 nuclear localization ...
Programmed cell death, in particular the intrinsic apoptotic pathway, has been shown to play a critical role in the shaping of tissues during embryonic development. The multi-BCL-2 Homology (BH) domain effectors of apoptosis, BAX, BAK, and BOK, are essential for cell killing in the intrinsic...
Haiyang Li ,Na Zheng &Shuli Zhao Browse broader subjects Cancer Browse Cancer microenvironment across other nature.com journals Previouspage page1 page2 page3 … page5 Nextpage Cell Death & Disease (Cell Death Dis)ISSN2041-4889(online)
Cell Death & Disease volume 5, page e1552 (2014)Cite this article 22k Accesses 53 Altmetric Metrics details Abstract In Slavic folklore, Koschei the Immortal was bony, thin and lean. Was his condition caused by severe calorie restriction (CR)? CR deactivates the target of rapamycin pathway ...
New insights into crosstalk between Nrf2 pathway and ferroptosis in lung disease Yonghu Chen Zhe Jiang Xuezheng Li Review ArticleOpen Access18 Nov 2024 NINJ1: Bridging lytic cell death and inflammation therapy Jinze Shen Ruixiu Chen Shiwei Duan ...
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