近日,哈尔滨医科大学寒地心血管病全国重点实验室杨宝峰院士团队在Science China Life Sciences发表了题为LncRNA CCRR maintains Ca2+homeostasis against myocardial infarction through the FTO-SERCA2a pathway的研究论文,揭示了lncRNA CCRR靶向FT...
近日,哈尔滨医科大学寒地心血管病全国重点实验室杨宝峰院士团队在Science China Life Sciences发表了题为LncRNA CCRR maintains Ca2+homeostasis against myocardial infarction through the FTO-SERCA2a pathway的研究论文,揭示了lncRNA CCRR靶...
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Although LWD1 has no recognizable DNA binding domain, it has 5 WD-repeats that constitute a propeller structure frequently considered a protein–protein interaction interface19. Therefore, LWD1 may be recruited to theCCA1promoter by interacting with transcription factors that remain to be identified. ...
our analysis revealed that higher expression of CCL24 is correlated with shorter OS. Jin et al. provided clues for the contribution of the CCL24 in hepatocellular carcinoma (HCC) malignancy through the RhoB-VEGFA-VEGFR2 angiogenesis pathway. They also indicated that upregulation of CCL24 in HCC...
[33] Li, Baijun, et al. "CCR9–CCL25 interaction suppresses apoptosis of lung cancer cells by activating the PI3K/Akt pathway." Medical Oncology 32 (2015): 1-9. [34] Niu, Yuxu, et al. "CCL25 promotes the migration and invasion of non‑small cell lung cancer cells by regulating ...
More importantly, M2 polarization increases PD-L2 expression in TAMs, resulting in immune evasion and tumor promotion through PD-1 signaling pathway. Conclusion This study highlights the role of CCL2-CCR2 axis in esophageal carcinogenesis. Our findings provide new insight into the mechanism of ...
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In one pathway, CCL2 induces MEK signaling through p42/44MAPK independently of Smad3 to promote cell survival. In addition, CCL2 signaling activates Smad3, which cooperates with the MEK-p42/44MAPK pathway to regulate cell motility and survival through RhoA-dependent mechanisms (Fig. 9). In ...