摘要: This article elucidates the clinical and pathological features of both ductal and lobular in-situ breast carcinoma. It quantifies the risk of invasive disease developing and outlines different treatment options with results as presently known....
Carcinoma, Pancreatic DuctalBreast NeoplasmsPancreatic NeoplasmsOvarian NeoplasmsUrinary Bladder NeoplasmsImmunohistochemistryNeoplasm InvasivenessMucin-1Invasive micropapillary carcinoma, a tumor with highly infiltrative characteristics is defined by a distinctive cleft formation around the neoplastic cell clusters which...
Ductal carcinoma in-situ: diagnosis and classification. Curr Diagn Pathol. 2004;10:204鈥 10. Kennedy M, Masterson AV, Kerin M, Flanagan F. Pathology and clinical relevance of radial scars: a review. J Clin Pathol. 2003;56:721鈥 4. CrossRef Lagios MD, Westdahl PR, Margolin FR, Roses...
C1 tumours (p = 1.8 ×10−14, Fisher’s Exact Test) (Fig.1b). Fig. 1: Derivation of prognostic clusters in TCGA SCC cohort. aConsensus clustering of 236 TCGA HPV + SCC patients produced an optimum 2 clusters (C1 and C2). Dark blue on the heat map represents patients (b...
Tertiary lymphoid structures are immune cell aggregates linked with cancer outcomes, but their interactions with tumour cell aggregates are unclear. Using nasopharyngeal carcinoma as a model, here we analyse single-cell transcriptomes of 343,829 cells fr
Immunohistochemistry facilitates the diagnosis of breast DCIS; myoepithelial and basal lamina markers are useful in differentiating microinvasive from in situ ductal carcinomas of the breast.doi:10.1007/s004280050332S. DamianiDepartment of Oncology, Section of Anatomic Pathology, University of Bologna, ...
The mean age of the patients (65 years) was slightly higher than that reported for pancreatic acinar cell carcinoma (58 years) and lower than the mean age for ductal adenocarcinoma (70 years).1,3 Three main questions Modern Pathology (2011) 24, 1620–1626 Pancreatic-type acinar cell ...
Tertiary lymphoid structures are immune cell aggregates linked with cancer outcomes, but their interactions with tumour cell aggregates are unclear. Using nasopharyngeal carcinoma as a model, here we analyse single-cell transcriptomes of 343,829 cells fr