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The c-Jun N-terminal protein kinase (JNK or SAPK1) signals downstream from the small GTPases Rac/Rho/Cdc42 that are known to regulate neurite outgrowth. JNK may also control alterations in sensory neuron phenotype induced by axotomy via activation of transcription factors such as AP-1 and ...
c-Jun氨基末端激酶（c-Jun N-terminal kinase，JNK）家族是促分裂活化蛋白激酶（MAPK）超家族的成员之一。JNK 信号通路可被细胞因子、生长因子、应激( 如电离辐射、渗透压、热休克和氧化损伤) 等多种因素激活，大量体外和活体实验均证实JNK与眼科疾病、慢性炎症、神经退行性病变、肿瘤、糖尿病、缺血再灌注损伤等临床...
C-Jun N-terminal Kinase 3 Deficiency Protects Neurons from Axotomy-induced Death in Vivo through Mechanisms Independent of c-Jun Phosphorylation. et al.C-Jun N-terminal kinase3deficiency protects neurons from axotomy-induced death in vivo through mechanisms independent of C-Jun phosphorylation....
a) producing a crystal of an unphosphorylated JNK3α (c-Jun N-terminal kinase 3α) molecule comprising amino acid residues 40-402 of SEQ ID NO:1 and a chemical entity; b) determining the three-dimensional atomic coordinates of amino acids Ile70, Gly71, Ser72, Gly73, Ala74, Gln75,...
Inhibitors of c-Jun N-Terminal Kinase 3The serine/threonine kinase c-Jun N-terminal kinase (JNK) 3 is implicated in the pathogenesis of various disorders ranging from neurodegenerative diseases to inflammation, metabolic disease,...doi:10.1007/7355_2020_98Koch, Pierre...
c-Jun氨基末端激酶(c-Jun N-terminal kinase,JNK)家族是促分裂活化蛋白激酶(MAPK)超家族的成员之一。JNK 信号通路可被细胞因子、生长因子、应激( 如电离辐射、渗透压、热休克和氧化损伤) 等多种因素激活,大…
中文名称 C-JUN N-TERMINAL KINASE 中文同义词 氨基末端激酶3重组兔单克隆抗体 英文名称 C-JUN N-TERMINAL KINASE 英文同义词 JNK3;C-JUN N-TERMINAL KINASE;SAPK1B/JNK3;c-Jun N-Terminal Kinase from rat muscle;JNK3, Stress-Activated Protein Kinase;Stress-Activated Protein Kinase;ANTI-MAPK10 antibody...
Both corresponded to the c-Jun NH2-terminal kinase (JNK) 3. In the present study, genomic cloning re- vealed that the JNK3 gene consists of 14 exons interrupted by 13 introns; its transcription-initiation site is within exon 3 and the termination codon lies in exon 14. Fluorescence in ...
c-Jun氨基末端激酶(c-Jun N-terminal kinase,JNK)家族是促分裂活化蛋白激酶(MAPK)超家族的成员之一。JNK 信号通路可被细胞因子、生长因子、应激( 如电离辐射、渗透压、热休克和氧化损伤) 等多种因素激活,大量体外和活体实验均证实JNK与眼科疾病、慢性炎症、神经退行性病变、肿瘤、糖尿病、缺血再灌注损伤等临床疾病...