K., Perchuk, B. S. & Laub, M. T. A DNA damage-induced, SOS-independent checkpoint regulates cell division in Caulobacter crescentus. PLoS Biol. 12, e1001977 (2014). Article Google Scholar Lund, V. A. et al. Molecular coordination of Staphylococcus aureus cell division. eLife 7, e3...
ly injected with 1 × 106GL261 cells.kKaplan-Meier analysis was performed on mouse models with intracranially implanted tumors derived from GL261 and treated with the indicated agents to assess survival (n = 3 mice per group).lRepresentative images of the luciferase signal from C57BL...
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医疗仪器滤光片 产品介绍: 医疗仪器滤光片指应用于酶标仪的340nm 405nm 450nm 510nm 546nm 578nm 630nm 670nm 700nm等半带宽19nm窄带滤光片组和45度分光镜。 针对医疗生化分析,荧光检测设备中物质发出的 的光谱进行处理,本司生产的额光学滤光片在这些系统中起到光隔离,光补偿,光激发,光显示与...
V.S. Lamzin, K.S. Wilson Automated refinement of protein models Acta Crystallogr. D Biol. Crystallogr., 49 (1993), pp. 129-147 Google Scholar [34] G.N. Murshudov,et al. REFMAC5 for the refinement of macromolecular crystal structures ...
应用:人核因子κ-Bp100亚基(NFKB2)酶联免疫(elisa)试剂盒可以在体外定量测定血浆、组织匀浆和其他生物液体中的人核因子κ-Bp100亚基(NFKB2)浓度. 检测方法:双抗夹心法 检测范围:0.313-20ng/ml 灵敏度 lt; 0.188ng/ml 标准曲线: 回收率:将一定含量的人核因子κ-Bp100亚基(NFKB2)掺入下表列出的样本中,...
Total RNA was prepared by homogenizing ten oocytes from each stage in 200 μl of buffer containing 100 mM Tris-HCl (pH 8.0), 150 mM NaCl, 12.5 mM EDTA, 1% SDS, 0.3 mg/ml tRNA, and 0.5 mg/ml proteinase K. After incubation for 1 hr at 50°C, the homogenates were adjusted to 250...
5H–K). Additionally, we validated the effects of migration and invasion for HK2 in FTO and ALKBH5 stable knockdown CRC cells. The results showed that silencing HK2 led to the attenuation of cell migration and invasion induced by FTO/ALKBH5 knockdown (Fig. 5L–N). In addition to knocking...
14. Bell JL, Wachter K, Muhleck B, Pazaitis N, Kohn M, Lederer M. et al. Insulin-like growth factor 2 mRNA-binding proteins (IGF2BPs): post-transcriptional drivers of cancer progression? Cell Mol Life Sci. 2013;70:2657-75 ...
We previously demonstrated that overexpression of RanBP9 led to enhanced Aβ generation in a variety of cell lines and primary neuronal cultures, and subsequently, we confirmed increased amyloid plaque burden in a mouse model of Alzheimer’s disease (AD)