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The activation of the Akt can promote the up-regulation of MDM2, a main negative regulators of P53, that may strengthen the degradation of P53 and inhibit its phosphorylation to prevent apoptosis and modulate the cell cycle [57]. Conclusion This study's findings indicated that SNKAF efficacy ...
Interestingly, HMGB1 levels were significantly elevated in HUVECs in the presence of GLU or NMDA (Fig.3A). To further validate whether HMGB1 is involved in NMDAR activation-regulated ferroptosis, we used HMGB1 siRNA to investigate the role of HMGB1 after NMDAR activation. Following knockdown of HM...
Here, we propose that miR-667-5p is involved in the DN process, and by targeting NLRC4, it inhibits NLRC4 inflammasome activation and inhibits the proliferation of renal tubular epithelial cells. However, more experiments are needed to confirm these findings in clinical samples. In addition, ...
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There is a correlation between stemness, autophagy and drug resistance [89]. In myeloma cells, PIWIL1 overexpression resulted in higher cell viability and resistance to bortezomib, dexamethasone, and doxorubicin in vitro due to the activation of autophagy and mitophagy. In line with that, PIWIL1 ...
The nucleotide-binding domain, leucine-rich-containing family, pyrin domain-containing-3 (NLRP3) inflammasome is another pathway that may mediate TMAO-induced renal fibrosis. In rat models of diabetic kidney disease, dietary supplementation with TMAO and choline were found to increase activation of NL...
Colon CSCs have many biological characteristics from other solid tumors such as self-renewal; abnormal activation of proliferating signaling pathways such as Notch and Hedgehog. It also mediates tumorigenicity and drug resistance [76]. Another study demonstrated that Vitk3 inhibited cell growth and ...
Exosomes derived from miR-181-5p-modified adipose-derived mesenchymal stem cells prevent liver fibrosis via autophagy activation. J Cell Mol Med. 2017;21(10):2491–502. https://doi.org/10.1111/jcmm.13170. Article CAS PubMed PubMed Central Google Scholar Deng S, Zhou X, Ge Z, Song Y,...
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