8.atherosclerosisplaque粥样硬化斑块;动脉粥样硬化;动脉粥样硬化斑块 9.Anti-atherosclerosis抗动脉粥样硬化 10.carotid arteryatherosclerosis颈动脉粥样硬化;动脉粥样硬化;颅外颈动脉粥样硬化;颈动脉粥样硬化组 用法例句 1. Gum problems increase the risk ofatherosclerosis. ...
Importantly, we did not observe any significant difference among these control mice with respect to atherosclerotic plaque formation. To generate IP3R1 LoxP-Cre mice, IP3R1 was flanked by LoxP sites and the EC-specific Cre mouse was bred with IP3R1fl/fl mice (iCDH5-Cre ERT2). The inducible...
Notably, the TG/HDL-C ratio is associated with carotid artery intima media thickness (cIMT) and carotid artery plaque [23], [24], [25], established proxies of subclinical atherosclerosis with predictive ability for future coronary heart disease (CHD) [26]. Moreover, it has been suggested ...
The biodegradable NPs were capable of reducing plaque development, necrotic core size, and lowering reactive oxygen species (ROS) in an atherosclerosis mouse model (ldlr−/− on a high fat diet) (Figure 5C). This study adds to the research underlining IL-10 as important in curbing the ...
Visfatin had a positive correlation with IMT ≥ 1.0 mm or plaque (p = 0.008) but resistin only correlated with plaque (p = 0.049). Visfatin predicted IMT ≥ 1.0 mm or plaque in patients on β-blocker monotherapy (p = 0.031). Visfatin lost its ability to predict subclinical atherosclerosis...
In mice with atherosclerosis, treatment with benzo-LXA4 (1R)-3a for 6 weeks, initiated 10 weeks after the onset of diabetes, resulted in a significant reduction in plaque development in the aortic arch [282]. In addition to lipoxins, efforts on the pharmacological application of SPMs have ...
Fetuin-A and atherosclerotic calcified plaque in patients with type 2 diabetes mellitus. Metabolism 2010, 59, 873–878. [CrossRef] [PubMed] 37. Hansson, G. Inflammation, atherosclerosis, and coronary artery disease. N. Engl. J. Med. 2005, 16, 1685–1695. [CrossRef] [PubMed] 38. Levi...
2017, 18, 781 2 of 10 effects from proteasome inhibition in early atherosclerosis are conceivable, since a dual role of the UPS in atherosclerotic plaque progression was postulated [1,8]. It was assumed that the UPS may be overreactive at an early stage, thereby driving the inflammatory ...
Introduction Atherosclerosis is regarded as a chronic inflammatory disease in response to injury of the arterial wall in which the inside stenosis is due to plaque formation [1]. Vascular inflammation stimulates the expression of adhesion molecules, such as intercellular adhesion molecule 1 (ICAM1), ...
It is possible that the "alternative" M2 milieu in the lesion microenvironment (in this case PVAT) promotes regression of atherosclerosis and/or plaque stabilization, as is evidenced by the reduced necrotic core size. Previous observations in humans support this notion, as epicardial adipose tissue...