Rad52抗体,Anti-Rad52产品信息: 中文名称:Rad52抗体,Anti-Rad52 英文名称:Anti-Rad52 别名: DNA repair protein RAD52; DNA repair protein RAD52 homolog; RAD 52; RAD52 homolog (S. cerevisiae); RAD52 homolog; Recombination protein RAD52; Rhabdomyosarcoma antigen MU RMS 40.23. ...
This gene product was shown to bind single-stranded DNA ends, and mediate the DNA-DNA interaction necessary for the annealing of complementary DNA strands. It was also found to interact with DNA recombination protein RAD51, which suggested its role in RAD51 related DNA recombination and repair....
It was also found to interact with DNA recombination protein RAD51, which suggested its role in RAD51 related DNA recombination and repair. [provided by RefSeq, Jul 2008] Also known as: DNA repair protein RAD52; DNA Anti-Rad52抗体repair protein RAD52 homolog; RAD 52; RAD52 homolog (S...
This mutation suppresses a broad spectrum of srs2螖 phenotypes in haploid cells, such as UV and 纬-ray sensitivities as well as synthetic lethality with replication and recombination mutants, while it does not significantly affect Rad52 functions in HR and DNA repair. Extensive analysis of the ...
The protein encoded by this gene is a member of the RAD51 protein family. RAD51 family members are highly similar to bacterial RecA and Saccharomyces cerevisiae Rad51, and are known to be involved in the homologous recombination and repair of DNA. This protein can interact with the ssDNA-bind...
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Nucleosomes wrap and compact DNA into chromatin, limiting DNA accessibility to the cellular machineries which require DNA as a template. Histones thereby play a central role in transcription regulation, DNA repair, DNA replication and chromosomal stability. DNA accessibility is regulated via a complex ...
As part of the alternative replication protein A complex, aRPA, binds single-stranded DNA and probably plays a role in DNA repair. Compared to the RPA2-containing, canonical RPA complex, may not support chromosomal DNA replication and cell cycle progression through S-phase. The aRPA may not ...
in ovarian cancer21, including aberrant expression of splicing factors such as SRSF3 and SFPQ22,23. Hyperactivation of MYC can lead to global upregulation of pre-mRNA levels and to aberrant splicing patterns in ovarian cancer24. However, knowledge of the role of splicing factors in the ...
The use of oncolytic viruses as an agent of combination therapy for cancer treatment also gained much attention in the recent past. This review focuses on the emerging role of oncolytic viruses as vital components of anti-cancer regimen presenting a new dimension in an ever-changing cancer ...