BioXCell热销产品—InVivoMAb anti-mouse IFNγ 2022-03-09 浏览次数:134 克隆号XMG1.2单克隆抗体能与小鼠干扰素γ(interferon gamma)反应,干扰素γ是一种20 kDa可溶性多效性细胞因子,是第二类干扰素的唯一成员。IFNγ主要由活化的淋巴细胞产生,包括T、B、NK细胞和ILCs。IFNγ发挥免疫调节、抗增殖、抗病毒和促炎...
InVivoMAb抗小鼠NK1.1/InVivoMAb anti-mouse NK1.1方案 PK136单克隆抗体与小鼠NK1.1(也称为CD161b/CD161c、KLRB1、NKR-P1A和Ly-55)反应。NK1.1是一种具有C型凝集素结构域的II型整合膜糖蛋白,由Klrb1c/NKR-P1C基因编码。NK1.1在NK细胞活化和分化、IFN-γ产生、细胞毒性颗粒释放中起作用,并被认为参与Th2细胞...
(2016). “Loss of IFN-gamma Pathway Genes in Tumor Cells as a Mechanism of Resistance to Anti-CTLA-4 Therapy.” Cell 167(2): 397-404 e399 in vivo CTLA-4 neutralization Bartkowiak, T., et al. (2015). “Unique potentia...
不要冷冻。 InVivoMAb抗小鼠NK1.1/InVivoMAb anti-mouse NK1.1文献参考: in vivo NK cell depletion Glasner, A., et al. (2018). "NKp46 Receptor-Mediated Interferon-gamma Production by Natural Killer Cells Increases Fibronectin 1 to Alter Tumor Architecture and Control Metastasis" Immunity 48(1):...
C(8)Mab-3: An Anti-Mouse CCR8 Monoclonal Antibody for Immunocytochemistry. PMID: 35377236 CCR8 Signaling via CCL1 Regulates Responses of Intestinal IFN-gamma Producing Innate Lymphoid CelIs and Protects From Experimental Colitis. PMID: 33613532 ...
CD4抗原是属于免疫球蛋白超家族的55 kDa细胞表面I型膜糖蛋白。CD4作为一种共受体,与T细胞受体(TCR)合作,与抗原呈递细胞(APC)显示的class II MHC分子相互作用。大多数胸腺细胞、大多数辅助性T细胞、NK-T细胞的一个亚群表达CD4,树突状细胞和巨噬细胞表达较少。...
InVivoMAb rat IgG2b isotype control, anti-keyhole limpet hemocyanin(货号:BE0090) 推荐抗体稀释液 InVivoPure™ pH 7.0 Dilution Buffer(货号:IP0070) 免疫原 CBA mouse thymocytes 应用 in vivo CD8+ T cell depletion/Western blot 产品形式 PBS , pH 7.0 Contains no stabilizers or preservatives ...
in vivo regulatory T cell depletionDeligne, C., et al. (2015). “Anti-CD20 therapy induces a memory Th1 response through the IFN-gamma/IL-12 axis and prevents protumor regulatory T-cell expansion in mice.” Leukemia 29(4): 947-957 ...
BioXcell 特色推荐——InVivoMAbanti-mouseCD4 一 产品背景 克隆号 GK1.5 单克隆抗体能与小鼠 CD4 反应。CD4 抗原是属于 免疫球蛋白超家族的 55 kDa 细胞表面 I 型膜糖蛋白。CD4 作为一种共 受体,与 T 细胞受体(TCR)合作,与抗原呈递细胞(APC)显示的 class II MHC 分子相互作用。大多数胸腺细胞、大多数辅助...
Dietary POA repaired gut mucosal barriers, reduced inflammatory cell infiltrations and expressions of TNF-α and IL-6, and improved efficacy of anti-TNF-α therapy in both acute and chronic IBD mouse models. Ex vivo treatment with POA in cultured inflamed colon tissues derived from Crohn's ...