上海玉博生物科技有限公司 主要致力于“Dystrophin (N-terminus) Vector-VP-D507”的生产销售。多年的“Dystrophin (N-terminus) Vector-VP-D507”生产与销售的经验,与各行业新老用户建立了稳定的合作关系,我公司经营的产品名称深受广大用户信赖。欢迎来电咨询或前来选购
指导意见：你的这种情况最 好是到三甲医院具体检查一下,根据具体的检查结果确定治疗的方案,不要忽视这种...
N Shiga,Y Takeshima,H Sakamoto,... - 《Journal of Clinical Investigation》 被引量: 195发表: 1997年 Disruption of the splicing enhancer sequence within exon 27 of the dystrophin gene by a nonsense mutation induces partial skipping of the exon and is respo... Shiga, N., Takeshima, Y., Sa...
英文别名: Dystrophin antibody [0.N.258] 中文名: 中文别名: CBNumber: CB95728513 分子式: 分子量: 0 MOL File: Mol file 化学性质 安全信息 用途 供应商 7 Dystrophin antibody [0.N.258]化学性质 安全信息Dystrophin antibody [0.N.258]性质、用途与生产工艺 Dystrophin antibody [0.N.258] 上下游产品...
Two actin-binding sites have been identified on human dystrophin by proton NMR spectroscopy of synthetic peptides corresponding to defined regions of the polypeptide sequence. These are Actin-Binding Site 1 (ABS1) located at residues 17–26 and Actin-Binding Site 2 (ABS2) in the region of res...
Biochemical and structural studies on the actin binding N-terminal domain of the dystrophin protein. 来自 ethos.bl.uk 喜欢 0 阅读量: 15 作者: FL Margaret 摘要: No abstract available 关键词: Muscular dystrophy Bacteria Protein crytals Biochemistry Molecular biology Cytology Genetics Microbiology ...
上海玉博生物科技有限公司 主要致力于“Mouse monoclonal [0.N.258] to Dystrophin - BSA and Azide free Abcam ab14452 ”的生产销售。多年的“Mouse monoclonal [0.N.258] to Dystrophin - BSA and Azide free Abcam ab14452 ”生产与销售的经验,与各行业新老用户建立
Myocardial infarction enterovirus dystrophin dystrophin-glycoprotein complex and gene polymorphisms in elderly patients Purpose The purpose of this work was to evaluate a role and mechanisms of active enterovirus infections (EVI), and genetic polymorphisms in the development of myocardial infarction (MI) in...
Dystrophin antibody [0.N.258] structure CAS No. Chemical Name: Dystrophin antibody [0.N.258] Synonyms Dystrophin antibody [0.N.258] CBNumber: CB95728513 Molecular Formula: Molecular Weight: 0 MDL Number: MOL File: Mol file Request For Quotation ...
Background Mutations in the DMD gene encoding dystrophin—a critical structural element in muscle cells—cause Duchenne muscular dystrophy (DMD), which is the most common fatal genetic disease. Clustered regularly interspaced short palindromic repeat (CRISPR)-mediated gene editing is a promising strategy...