which can deteriorate into acute respiratory distress syndrome. The primary etiology may be attributed to cytokine storm, which is triggered by the excessive release of proinflammatory cytokines
Our results need to be interpreted with caution considering the potential for residual confounders, and more well-designed studies that control the clinical confounders are necessary to confirm our findings.doi:10.1002/clc.23421John Wiley & Sons, LtdClinical Cardiology...
3.4. Bradykinin Finally, the RAS system, unbalanced in COVID-19 due to an excess of angiotensin II and a lack of ACE2, leads to an increase in bradykinin, an important mediator of acute inflammation (especially exudation and pain) [29] involved in vasodilation, plasma ex- travasation, ...
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